VITAMIN-C STABILIZATION AS A CONSEQUENCE OF THE PLASMA-MEMBRANE REDOX SYSTEM

被引：6

作者：

RODRIGUEZAGUILERA, JC ^{[1
]}

NAVARRO, F ^{[1
]}

ARROYO, A ^{[1
]}

ALCAIN, FJ ^{[1
]}

VILLALBA, JM ^{[1
]}

NAVAS, P ^{[1
]}

机构：

[1] UNIV CORDOBA,FAC CIENCIAS,DEPT BIOL CELULAR,AVDA SAN ALBERTO MAGNO S-N,E-14004 CORDOBA,SPAIN

来源：

PROTOPLASMA | 1995年 / 184卷 / 1-4期

关键词：

ASCORBATE STABILIZATION; ASCORBATE FREE RADICAL; PLASMA MEMBRANE REDOX SYSTEM; HL-60; CELLS; CYCLIC AMP;

D O I：

10.1007/BF01276925

中图分类号：

Q94 [植物学];

学科分类号：

071001 ;

摘要：

Ascorbate is stabilized in the presence of HL-60 cells. Our results showed that cAMP derivatives and agents that increase cAMP stimulate the ability of HL-60 cells to stabilize ascorbate. On the other hand, tunicamycin, a glycosilation-interfering agent, inhibited this ability. The ascorbate stabilization in the presence of HL-60 cells has been questioned as a simple chemical effect. Further properties and controls about the enzymatic nature of this stabilization are described and discussed. This data, together with hormonal regulation, support the hypothesis that an enzymatic redox system located at the plasma membrane is responsible of the extracellular ascorbate stabilization by HL-60 cells.

引用

页码：229 / 232

页数：4

共 22 条

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