O-2 FREE-RADICALS - CAUSE OF ISCHEMIA-REPERFUSION INJURY TO CARDIAC NA+-K+-ATPASE

被引：235

作者：

KIM, MS ^{[1
]}

AKERA, T ^{[1
]}

机构：

[1] MICHIGAN STATE UNIV,DEPT PHARMACOL & TOXICOL,B445 LIFE SCI BLDG,E LANSING,MI 48824

来源：

AMERICAN JOURNAL OF PHYSIOLOGY | 1987年 / 252卷 / 02期

关键词：

D O I：

10.1152/ajpheart.1987.252.2.H252

中图分类号：

Q4 [生理学];

学科分类号：

071003 ;

摘要：

引用

页码：H252 / H257

页数：6

共 31 条

[1] SIMPLE METHOD FOR DETERMINATION OF AFFINITY AND BINDING-SITE CONCENTRATION IN RECEPTOR-BINDING STUDIES [J].

AKERA, T ;

CHENG, VJK .

BIOCHIMICA ET BIOPHYSICA ACTA, 1977, 470 (03) :412-423

[2]

BEAUCHAMP C, 1970, J BIOL CHEM, V245, P4641

[3] ISCHEMIA-INDUCED ALTERATIONS IN MYOCARDIAL (NA+ + K+)-ATPASE AND CARDIAC GLYCOSIDE BINDING [J].

BELLER, GA ;

CONROY, J ;

SMITH, TW .

JOURNAL OF CLINICAL INVESTIGATION, 1976, 57 (02) :341-350

[4] ROLE OF NADPH-CYTOCHROME B5 REDUCTASE IN MICROSOMAL LIPID PEROXIDATION [J].

BIDLACK, WR ;

OKITA, RT ;

HOCHSTEIN, P .

BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1973, 53 (02) :459-465

[5] STUDIES ON SODIUM-POTASSIUM-ACTIVATED ADENOSINE TRIPHOSPHATASE .1. QUANTITATIVE DISTRIBUTION IN SEVERAL TISSUES OF CAT [J].

BONTING, SL ;

HAWKINS, NM ;

SIMON, KA .

ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS, 1961, 95 (03) :416-&

[6]

BRACHFELD N, 1974, B NEW YORK ACAD MED, V50, P261

[7] POSSIBLE MECHANISMS OF VENTRICULAR ARRHYTHMIAS ELICITED BY ISCHEMIA FOLLOWED BY REPERFUSION - STUDIES ON ISOLATED CANINE VENTRICULAR TISSUES [J].

FERRIER, GR ;

MOFFAT, MP ;

LUKAS, A .

CIRCULATION RESEARCH, 1985, 56 (02) :184-194

[8]

FREEMAN BA, 1982, LAB INVEST, V47, P412

[9]

Fridovich I, 1979, Adv Neurol, V26, P255

[10]

GUADUEL Y, 1984, J MOL CELL CARDIOL, V16, P459

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