INTERLEUKIN-1-BETA INDUCED CERAMIDE AND DIACYLGLYCEROL GENERATION DOES NOT LEAD TO NF-KAPPA-B ACTIVATION IN RINM5F CELLS

被引：0

作者：

WELSH, N ^{[1
]}

机构：

[1] UNIV UPPSALA,DEPT MED CELL BIOL,S-75105 UPPSALA,SWEDEN

来源：

DIABETOLOGIA | 1995年 / 38卷

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D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：A82 / A82

页数：1

共 37 条

[1] INTERLEUKIN-1-BETA INDUCED ACTIVATION OF NF-KAPPA-B IN INSULIN-PRODUCING RINM5F CELLS IS PREVENTED BY THE PROTEASE INHIBITOR N-ALPHA-P-TOSYL-L-LYSINE CHLOROMETHYLKETONE
SALDEEN, J
WELSH, N
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1994, 203 (01) : 149 - 155
[2] Interleukin-1 beta-mediated inhibition of arginase in RINm5F cells
Cunningham, JM
Mabley, JG
Green, IC
CYTOKINE, 1997, 9 (08) : 570 - 576
[3] IL-1-BETA INDUCES THE EXPRESSION OF NITRIC-OXIDE SYNTHASE BY RAT ISLETS OF LANGERHANS AND RINM5F CELLS - EVIDENCE FOR THE INVOLVEMENT OF NF-KAPPA-B IN THE SIGNALING MECHANISM
KWON, G
RODI, CP
SULLIVAN, PM
MCDANIEL, ML
DIABETES, 1994, 43 : A234 - A234
[4] Interleukin-1 beta-induced ceramide and diacylglycerol generation may lead to activation of the c-Jun NH2-terminal kinase and the transcription factor ATF2 in the insulin-producing cell line RINm5F
Welsh, N
JOURNAL OF BIOLOGICAL CHEMISTRY, 1996, 271 (14) : 8307 - 8312
[5] CHARACTERIZATION OF A FUNCTIONAL NF-KAPPA-B SITE IN THE HUMAN INTERLEUKIN-1-BETA PROMOTER - EVIDENCE FOR A POSITIVE AUTOREGULATORY LOOP
HISCOTT, J
MAROIS, J
GAROUFALIS, J
DADDARIO, M
ROULSTON, A
KWAN, I
PEPIN, N
LACOSTE, J
NGUYEN, H
BENSI, G
FENTON, M
MOLECULAR AND CELLULAR BIOLOGY, 1993, 13 (10) : 6231 - 6240
[6] INTERLEUKIN-1 BETA-INDUCED NITRIC-OXIDE PRODUCTION ACTIVATES APOPTOSIS IN PANCREATIC RINM5F CELLS
ANKARCRONA, M
DYPBUKT, JM
BRUNE, B
NICOTERA, P
EXPERIMENTAL CELL RESEARCH, 1994, 213 (01) : 172 - 177
[7] Celastrol ameliorates cytokine toxicity and pro-inflammatory immune responses by suppressing NF-κB activation in RINm5F beta cells
Ju, Sung Mi
Youn, Gi Soo
Cho, Yoon Shin
Choi, Soo Young
Park, Jinseu
BMB REPORTS, 2015, 48 (03) : 172 - 177
[8] TUMOR-NECROSIS-FACTOR AND INTERLEUKIN-1 LEAD TO PHOSPHORYLATION AND LOSS OF I-KAPPA-B-ALPHA - A MECHANISM FOR NF-KAPPA-B ACTIVATION
BEG, AA
FINCO, TS
NANTERMET, PV
BALDWIN, AS
MOLECULAR AND CELLULAR BIOLOGY, 1993, 13 (06) : 3301 - 3310
[9] Inhibitory effects of epicatechin on interleukin-1β-induced inducible nitric oxide synthase expression in RINm5F cells and rat pancreatic islets by down-regulation of NF-κB activation
Kim, MJ
Ryu, GR
Kang, JH
Sim, SS
Min, DS
Rhie, DJ
Yoon, SH
Hahn, SJ
Jeong, IK
Hong, KJ
Kim, MS
Jo, YH
BIOCHEMICAL PHARMACOLOGY, 2004, 68 (09) : 1775 - 1785
[10] THE SPONTANEOUS EXPRESSION OF INTERLEUKIN-1-BETA AND INTERLEUKIN-6 IS ASSOCIATED WITH SPONTANEOUS EXPRESSION OF AP-1 AND NF-KAPPA-B TRANSCRIPTION FACTOR IN ACUTE MYELOBLASTIC-LEUKEMIA CELLS
DOKTER, WHA
TUYT, L
SIERDSEMA, SJ
ESSELINK, MT
VELLENGA, E
LEUKEMIA, 1995, 9 (03) : 425 - 432

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