THROMBIN RECEPTOR-ACTIVATING PEPTIDES (TRAPS) ELICIT CALCIUM TRANSIENTS IN UMR-106 CELLS

被引：0

|

作者：

SANDERS, J

ANDERSEN, T

STERN, P

机构：

[1] NORTHWESTERN UNIV,CHICAGO,IL 60611

[2] ALBANY MED COLL,ALBANY,NY 12208

来源：

JOURNAL OF BONE AND MINERAL RESEARCH | 1993年 / 8卷

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

收藏

页码：S186 / S186

页数：1

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[41] Possible regulation of epidermal growth factor-receptor tyrosine autophosphorylation by calcium and G proteins chemically permeabilized rat UMR106 cells
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[43] Insulin-like growth factor I suppresses parathyroid hormone (PTH)/PTH-related protein receptor expression via a mitogen-activated protein kinase pathway in UMR-106 osteoblast-like cells
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ENDOCRINOLOGY, 1999, 140 (02) : 871 - 879
[44] Parathyroid hormone (PTH) down-regulates PTH/PTH-related protein receptor gene expression in UMR-106 osteoblast-like cells via a 3′,5′-cyclic adenosine monophosphate-dependent, protein kinase A-independent pathway
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[45] Insulin-like growth factor I suppresses parathyroid hormone (PTH)/PTH-related protein receptor expression via a mitogen-activated protein kinase pathway in UMR-106 osteoblast-like cells (vol 140, pg 871, 1999)
Kawane
Horiuchi
ENDOCRINOLOGY, 1999, 140 (03) : 1491 - 1491
[46] Protease-activated receptors in primary human brain microvascular endothelial cells: Differential calcium signaling induced by thrombin and protease-activated receptor-1 activating peptide
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[47] TRANSMEMBRANE SIGNALING DEFECTS IN WISKOTT-ALDRICH SYNDROME PATIENT B-CELLS REVEAL A DICHOTOMY IN THE SIGNALING PATHWAYS WHEREBY PLATELET-ACTIVATING-FACTOR AND SURFACE IG RECEPTOR ENGAGEMENT ELICIT INCREASES IN CYTOSOLIC CALCIUM
SIMINOVITCH, KA
SIMON, HU
MILLS, GB
JOURNAL OF CELLULAR BIOCHEMISTRY, 1993, : 185 - 185

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