EXCESSIVE CELLULAR ACIDOSIS - AN IMPORTANT MECHANISM OF NEURONAL DAMAGE IN THE BRAIN

被引：208

作者：

REHNCRONA, S ^{[1
]}

ROSEN, I ^{[1
]}

SIESJO, BK ^{[1
]}

机构：

[1] UNIV LUND HOSP,DEPT NEUROSURG,S-22185 LUND,SWEDEN

来源：

ACTA PHYSIOLOGICA SCANDINAVICA | 1980年 / 110卷 / 04期

关键词：

D O I：

10.1111/j.1748-1716.1980.tb06692.x

中图分类号：

Q4 [生理学];

学科分类号：

071003 ;

摘要：

引用

页码：435 / 437

页数：3

共 7 条

[1] REVERSIBILITY OF ISCHEMIC BRAIN-DAMAGE [J].

HOSSMANN, KA ;

KLEIHUES, P .

ARCHIVES OF NEUROLOGY, 1973, 29 (06) :375-384

[2] INFLUENCE OF TISSUE ACIDOSIS UPON RESTITUTION OF BRAIN ENERGY METABOLISM FOLLOWING TOTAL ISCHEMIA [J].

LJUNGGREN, B ;

NORBERG, K ;

SIESJO, BK .

BRAIN RESEARCH, 1974, 77 (02) :173-186

[3]

MYERS RE, 1979, CEREBRAL HYPOXIA ITS, P195

[4] EFFECTS OF PHENOBARBITAL IN CEREBRAL ISCHEMIA .2. RESTITUTION OF CEREBRAL ENERGY-STATE, AS WELL AS OF GLYCOLYTIC METABOLITES, CITRIC-ACID CYCLE INTERMEDIATES AND ASSOCIATED AMINO-ACIDS AFTER PRONOUNCED INCOMPLETE ISCHEMIA [J].

NORDSTROM, CH ;

REHNCRONA, S ;

SIESJO, BK .

STROKE, 1978, 9 (04) :335-343

[5] INFLUENCE OF COMPLETE AND PRONOUNCED INCOMPLETE CEREBRAL-ISCHEMIA AND SUBSEQUENT RECIRCULATION ON CORTICAL CONCENTRATIONS OF OXIDIZED AND REDUCED GLUTATHIONE IN THE RAT [J].

REHNCRONA, S ;

FOLBERGROVA, J ;

SMITH, DS ;

SIESJO, BK .

JOURNAL OF NEUROCHEMISTRY, 1980, 34 (03) :477-486

[6]

REHNCRONA S, J NEUROCHEM

[7] INCOMPLETE VERSUS COMPLETE CEREBRAL-ISCHEMIA - IMPROVED OUTCOME WITH A MINIMAL BLOOD-FLOW [J].

STEEN, PA ;

MICHENFELDER, JD ;

MILDE, JH .

ANNALS OF NEUROLOGY, 1979, 6 (05) :389-398

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