INTERFERON BETA-1, AN INTERMEDIATE IN THE TUMOR-NECROSIS-FACTOR ALPHA-INDUCED INCREASED MHC CLASS-I EXPRESSION AND AN AUTOCRINE REGULATOR OF THE CONSTITUTIVE MHC CLASS-I EXPRESSION

被引：36

作者：

LEEUWENBERG, JFM ^{[1
]}

VANDAMME, J ^{[1
]}

JEUNHOMME, GMAA ^{[1
]}

BUURMAN, WA ^{[1
]}

机构：

[1] CATHOLIC UNIV LEUVEN,REGA INST MED RES,B-3000 LOUVAIN,BELGIUM

来源：

JOURNAL OF EXPERIMENTAL MEDICINE | 1987年 / 166卷 / 04期

关键词：

D O I：

10.1084/jem.166.4.1180

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：1180 / 1185

页数：6

共 13 条

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[4] SELECTIVE REJECTION OF H-2-DEFICIENT LYMPHOMA VARIANTS SUGGESTS ALTERNATIVE IMMUNE DEFENSE STRATEGY [J].

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[6] INDUCTION OF INTERFERON-BETA-2 BY TUMOR-NECROSIS-FACTOR - A HOMEOSTATIC MECHANISM IN THE CONTROL OF CELL-PROLIFERATION [J].

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[9] IDENTIFICATION OF THE HUMAN 26-KD PROTEIN, INTERFERON-BETA-2 (IFN-BETA-2), AS A B-CELL HYBRIDOMA PLASMACYTOMA GROWTH-FACTOR INDUCED BY INTERLEUKIN-1 AND TUMOR-NECROSIS-FACTOR [J].

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[10]

VANDAMME J, 1987, IN PRESS J IMMUNOL

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