TISSUE HYPOXIA - CLINICAL AND BIOCHEMICAL ASPECTS

[1] DOES SLOW-REACTING SUBSTANCE OF ANAPHYLAXIS MEDIATE HYPOXIC PULMONARY VASOCONSTRICTION [J]. AMERICAN REVIEW OF RESPIRATORY DISEASE, 1983, 127 (05): : 566 - 571

[2] ROLE OF GLUTAMATE AS THE CENTRAL NEUROTRANSMITTER IN THE HYPOXIC VENTILATORY RESPONSE [J]. JOURNAL OF APPLIED PHYSIOLOGY, 1992, 72 (04) : 1480 - 1487

[3] ANTEZANA AM, 1992, INT J SPORTS MED S1, V13, P96

[4] A BRIEF PERIOD OF HYPOXIA CAUSES PROTEOLYSIS OF CYTOSKELETAL PROTEINS IN HIPPOCAMPAL SLICES [J]. BRAIN RESEARCH, 1991, 555 (02) : 276 - 280

[5] HEMIN - A POSSIBLE PHYSIOLOGICAL MEDIATOR OF LOW-DENSITY-LIPOPROTEIN OXIDATION AND ENDOTHELIAL INJURY [J]. ARTERIOSCLEROSIS AND THROMBOSIS, 1991, 11 (06): : 1700 - 1711

[6] BERGOFSKY H, 1986, ABNORMAL PULMONARY C

[7] BHAT GB, 1992, AM J PHYS LUNG CELL, V262, P606

[8] HYPOXIA AS A RISK FACTOR FOR DOXORUBICIN-INDUCED CARDIOTOXICITY - A NMR EVALUATION [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1989, 163 (02) : 682 - 688

[9] ELECTROCHEMICAL MONITORING OF BRAIN ASCORBIC-ACID CHANGES ASSOCIATED WITH HYPOXIA, SPREADING DEPRESSION, AND SEIZURE ACTIVITY [J]. NEUROCHEMICAL RESEARCH, 1992, 17 (01) : 23 - 27

[10] CHANG SW, 1992, AM J PHYS LUNG CELL, V263, P446

[1] DOES SLOW-REACTING SUBSTANCE OF ANAPHYLAXIS MEDIATE HYPOXIC PULMONARY VASOCONSTRICTION [J]. AMERICAN REVIEW OF RESPIRATORY DISEASE, 1983, 127 (05): : 566 - 571

[2] ROLE OF GLUTAMATE AS THE CENTRAL NEUROTRANSMITTER IN THE HYPOXIC VENTILATORY RESPONSE [J]. JOURNAL OF APPLIED PHYSIOLOGY, 1992, 72 (04) : 1480 - 1487

[3] ANTEZANA AM, 1992, INT J SPORTS MED S1, V13, P96

[4] A BRIEF PERIOD OF HYPOXIA CAUSES PROTEOLYSIS OF CYTOSKELETAL PROTEINS IN HIPPOCAMPAL SLICES [J]. BRAIN RESEARCH, 1991, 555 (02) : 276 - 280

[5] HEMIN - A POSSIBLE PHYSIOLOGICAL MEDIATOR OF LOW-DENSITY-LIPOPROTEIN OXIDATION AND ENDOTHELIAL INJURY [J]. ARTERIOSCLEROSIS AND THROMBOSIS, 1991, 11 (06): : 1700 - 1711

[6] BERGOFSKY H, 1986, ABNORMAL PULMONARY C

[7] BHAT GB, 1992, AM J PHYS LUNG CELL, V262, P606

[8] HYPOXIA AS A RISK FACTOR FOR DOXORUBICIN-INDUCED CARDIOTOXICITY - A NMR EVALUATION [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 1989, 163 (02) : 682 - 688

[9] ELECTROCHEMICAL MONITORING OF BRAIN ASCORBIC-ACID CHANGES ASSOCIATED WITH HYPOXIA, SPREADING DEPRESSION, AND SEIZURE ACTIVITY [J]. NEUROCHEMICAL RESEARCH, 1992, 17 (01) : 23 - 27

[10] CHANG SW, 1992, AM J PHYS LUNG CELL, V263, P446