Reaching out for the other end with p53-binding protein 1

被引：2

作者：

van Gent, Dik C. ^{[1
]}

机构：

[1] Erasmus MC, Dept Cell Biol & Genet, Rotterdam, Netherlands

来源：

TRENDS IN BIOCHEMICAL SCIENCES | 2009年 / 34卷 / 05期

关键词：

CLASS-SWITCH RECOMBINATION; DOUBLE-STRAND BREAKS; DNA-DAMAGE; 53BP1; REPAIR; MDC1; ATM; STABILITY; TELOMERES; P53;

D O I：

10.1016/j.tibs.2009.01.009

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Correct joining of broken chromosomes is indispensable to prevent cell death, ageing and cancer. Two recent papers highlight the importance of p53-binding protein 1 (53BP1) in connecting distant sites in T-cell receptor loci and dysfunctional telomeres. These studies refine our understanding of the specific subset of DNA double-strand breaks that require the accessory end-joining factor 53BP1.

引用

页码：226 / 229

页数：4

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