MITOCHONDRIAL CALCIUM HANDLING AND OXIDATIVE STRESS

[1] THE ENTRAPMENT OF THE CA-2+ INDICATOR ARSENAZO-III IN THE MATRIX SPACE OF RAT-LIVER MITOCHONDRIA BY PERMEABILIZATION AND RESEALING - NA+-DEPENDENT AND NA+-INDEPENDENT EFFLUXES OF CA-2+ IN ARSENAZO-III-LOADED MITOCHONDRIA [J]. BIOCHEMICAL JOURNAL, 1986, 239 (01) : 31 - 40

[2] BEATRICE MC, 1980, J BIOL CHEM, V255, P8663

[3] BERGSMA DJ, 1991, J BIOL CHEM, V266, P23204

[4] CYCLOSPORINE-A PROTECTS HEPATOCYTES SUBJECTED TO HIGH CA-2+ AND OXIDATIVE STRESS [J]. FEBS LETTERS, 1992, 304 (2-3) : 192 - 194

[5] BROEKEMEIER KM, 1989, J BIOL CHEM, V264, P7826

[6] CELL-DEATH BY APOPTOSIS AND ITS PROTECTIVE ROLE AGAINST DISEASE [J]. TRENDS IN PHARMACOLOGICAL SCIENCES, 1992, 13 (06) : 245 - 251

[7] THE INFLUENCE OF NUPERCAINE ON CA-2+ TRANSPORT BY RAT-LIVER AND EHRLICH ASCITES CELL MITOCHONDRIA [J]. FEBS LETTERS, 1982, 144 (02) : 279 - 282

[8] PURIFICATION AND N-TERMINAL SEQUENCING OF PEPTIDYL-PROLYL CIS-TRANS-ISOMERASE FROM RAT-LIVER MITOCHONDRIAL MATRIX REVEALS THE EXISTENCE OF A DISTINCT MITOCHONDRIAL CYCLOPHILIN [J]. BIOCHEMICAL JOURNAL, 1992, 284 : 381 - 385

[9] EVIDENCE FOR THE PRESENCE OF A REVERSIBLE CA-2+-DEPENDENT PORE ACTIVATED BY OXIDATIVE STRESS IN HEART-MITOCHONDRIA [J]. BIOCHEMICAL JOURNAL, 1987, 245 (03) : 915 - 918

[10] KINETIC EVIDENCE FOR A HEART MITOCHONDRIAL PORE ACTIVATED BY CA-2+, INORGANIC-PHOSPHATE AND OXIDATIVE STRESS - A POTENTIAL MECHANISM FOR MITOCHONDRIAL DYSFUNCTION DURING CELLULAR CA-2+ OVERLOAD [J]. EUROPEAN JOURNAL OF BIOCHEMISTRY, 1988, 178 (02): : 489 - 501

[1] THE ENTRAPMENT OF THE CA-2+ INDICATOR ARSENAZO-III IN THE MATRIX SPACE OF RAT-LIVER MITOCHONDRIA BY PERMEABILIZATION AND RESEALING - NA+-DEPENDENT AND NA+-INDEPENDENT EFFLUXES OF CA-2+ IN ARSENAZO-III-LOADED MITOCHONDRIA [J]. BIOCHEMICAL JOURNAL, 1986, 239 (01) : 31 - 40

[2] BEATRICE MC, 1980, J BIOL CHEM, V255, P8663

[3] BERGSMA DJ, 1991, J BIOL CHEM, V266, P23204

[4] CYCLOSPORINE-A PROTECTS HEPATOCYTES SUBJECTED TO HIGH CA-2+ AND OXIDATIVE STRESS [J]. FEBS LETTERS, 1992, 304 (2-3) : 192 - 194

[5] BROEKEMEIER KM, 1989, J BIOL CHEM, V264, P7826

[6] CELL-DEATH BY APOPTOSIS AND ITS PROTECTIVE ROLE AGAINST DISEASE [J]. TRENDS IN PHARMACOLOGICAL SCIENCES, 1992, 13 (06) : 245 - 251

[7] THE INFLUENCE OF NUPERCAINE ON CA-2+ TRANSPORT BY RAT-LIVER AND EHRLICH ASCITES CELL MITOCHONDRIA [J]. FEBS LETTERS, 1982, 144 (02) : 279 - 282

[8] PURIFICATION AND N-TERMINAL SEQUENCING OF PEPTIDYL-PROLYL CIS-TRANS-ISOMERASE FROM RAT-LIVER MITOCHONDRIAL MATRIX REVEALS THE EXISTENCE OF A DISTINCT MITOCHONDRIAL CYCLOPHILIN [J]. BIOCHEMICAL JOURNAL, 1992, 284 : 381 - 385

[9] EVIDENCE FOR THE PRESENCE OF A REVERSIBLE CA-2+-DEPENDENT PORE ACTIVATED BY OXIDATIVE STRESS IN HEART-MITOCHONDRIA [J]. BIOCHEMICAL JOURNAL, 1987, 245 (03) : 915 - 918

[10] KINETIC EVIDENCE FOR A HEART MITOCHONDRIAL PORE ACTIVATED BY CA-2+, INORGANIC-PHOSPHATE AND OXIDATIVE STRESS - A POTENTIAL MECHANISM FOR MITOCHONDRIAL DYSFUNCTION DURING CELLULAR CA-2+ OVERLOAD [J]. EUROPEAN JOURNAL OF BIOCHEMISTRY, 1988, 178 (02): : 489 - 501