MAINTENANCE OF DNA METHYLATION LEVEL IN SV40-INFECTED HUMAN-FIBROBLASTS DURING THEIR INVITRO LIMITED PROLIFERATIVE LIFE-SPAN

被引：22

作者：

MATSUMURA, T ^{[1
]}

HUNTER, JL ^{[1
]}

FAROOQ, M ^{[1
]}

HOLLIDAY, R ^{[1
]}

机构：

[1] NATL INST MED RES,DIV GENET,LONDON NW7 1AA,ENGLAND

来源：

EXPERIMENTAL CELL RESEARCH | 1989年 / 184卷 / 01期

关键词：

D O I：

10.1016/0014-4827(89)90373-X

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

引用

页码：148 / 157

页数：10

共 19 条

[11] MAINTENANCE OF MITOCHONDRIAL-DNA GENOMES DURING INVITRO AGING OF CULTURED HUMAN-FIBROBLASTS
REIS, RJS
GOLDSTEIN, S
CLINICAL RESEARCH, 1981, 29 (05): : A862 - A862
[12] DIFFERENTIAL EXTRACELLULAR-MATRIX GENE-EXPRESSION BY FIBROBLASTS DURING THEIR PROLIFERATIVE LIFE-SPAN INVITRO AND AT SENESCENCE
CHOI, AMK
OLSEN, DR
COOK, KG
DEAMOND, SF
UITTO, J
BRUCE, SA
JOURNAL OF CELLULAR PHYSIOLOGY, 1992, 151 (01) : 147 - 155
[13] SYNTHESIS OF NUCLEAR MATRIX COMPONENTS DURING THE INVITRO LIFE-SPAN OF HUMAN-DIPLOID FIBROBLASTS
DELLORCO, RT
WHITTLE, WL
MOLECULAR BIOLOGY OF THE CELL, 1992, 3 : A326 - A326
[14] C-HA-RAS-1 PROTO-ONCOGENE AMPLIFICATION AND OVEREXPRESSION DURING THE LIMITED REPLICATIVE LIFE-SPAN OF NORMAL HUMAN-FIBROBLASTS
SRIVASTAVA, A
NORRIS, JS
REIS, RJS
GOLDSTEIN, S
JOURNAL OF BIOLOGICAL CHEMISTRY, 1985, 260 (10) : 6404 - 6409
[15] INCREASE IN EXPRESSION LEVELS OF INTERFERON-INDUCIBLE GENES IN SENESCENT HUMAN-DIPLOID FIBROBLASTS AND IN SV40-TRANSFORMED HUMAN FIBROBLASTS WITH EXTENDED LIFE-SPAN
TAHARA, H
KAMADA, K
SATO, E
TSUYAMA, N
KIM, JK
HARA, E
ODA, K
IDE, T
ONCOGENE, 1995, 11 (06) : 1125 - 1132
[16] LIFE-SPAN ELONGATION OF WERNERS SYNDROME FIBROBLASTS BY CO-CULTURE WITH ORIGIN-DEFECTIVE SV40 DNA TRANSFORMED-CELLS
OHNO, T
YAMAGUCHI, N
HUMAN GENETICS, 1984, 68 (03) : 209 - 210
[17] SV40-TRANSFORMED NORMAL AND DNA-REPAIR-DEFICIENT HUMAN-FIBROBLASTS CAN BE TRANSFECTED WITH HIGH-FREQUENCY BUT RETAIN ONLY LIMITED AMOUNTS OF INTEGRATED DNA
MAYNE, LV
JONES, T
DEAN, SW
HARCOURT, SA
LOWE, JE
PRIESTLEY, A
STEINGRIMSDOTTIR, H
SYKES, H
GREEN, MHL
LEHMANN, AR
GENE, 1988, 66 (01) : 65 - 76
[18] INVITRO ESTABLISHMENT OF HUMAN-FIBROBLASTS OF LYSOSOMAL DISEASES, GM1-GANGLIOSIDOSIS AND SANDHOFF DISEASE, BY TRANSFORMATION WITH ORIGIN-MINUS SV40 DNA
MOMOI, T
FURUYA, T
SUZUKI, Y
SATO, H
YAMAGUCHI, N
BIOSCIENCE REPORTS, 1985, 5 (03) : 267 - 273
[19] Critical telomere shortening regulated by the ataxia-telangiectasia gene acts as a DNA damage signal leading to activation of p53 protein and limited life-span of human diploid fibroblasts. A review
Vaziri, H
BIOCHEMISTRY-MOSCOW, 1997, 62 (11) : 1306 - 1310

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