LEAF SENESCENCE IN A NONYELLOWING MUTANT OF FESTUCA-PRATENSIS - METABOLISM OF CYTOCHROME-F

被引:22
作者
DAVIES, TGE [1 ]
THOMAS, H [1 ]
THOMAS, BJ [1 ]
ROGERS, LJ [1 ]
机构
[1] UNIV COLL ABERYSTWYTH,DEPT BIOCHEM,ABERYSTWYTH SY23 3DD,DYFED,WALES
关键词
D O I
10.1104/pp.93.2.588
中图分类号
Q94 [植物学];
学科分类号
071001 ;
摘要
In a mutant genotype of Festuca pratensis Huds., net degradation of a number of thylakoid membrane proteins during senescence is impaired. Previous studies have suggested that the highly hydrophobic intrinsic chlorophyll-binding proteins were the definitive subjects of the metabolic lesion. In the present study we find that cytochrome f, as determined by haem-staining, Western blotting, enzyme-linked immunosorbent assay, and immunogold electron microscopy, is also abnormally stable in the mutant. The structural feature common to all the proteins in the mutant so far recognized to be abnormally stable is possession of a tetrapyrrole prosthetic group. It is suggested that degradation of chlorophyll and haem may regulate degradation of the associated apoproteins, and hence has an important role to play in membrane protein turnover and in mobilisation of amino acids during chloroplast disassembly.
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收藏
页码:588 / 595
页数:8
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