ORGAN-SPECIFIC AUTOIMMUNITY

[1] PREDICTIVE MEDICINE IN AUTOIMMUNE-DISEASES - FROM THE IDENTIFICATION OF GENETIC PREDISPOSITION AND ENVIRONMENTAL INFLUENCE TO PRECOCIOUS IMMUNOTHERAPY [J]. CLINICAL IMMUNOLOGY AND IMMUNOPATHOLOGY, 1994, 72 (02): : 156 - 161

[2] T-CELL-MEDIATED INHIBITION OF THE TRANSFER OF AUTOIMMUNE DIABETES IN NOD MICE [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1989, 169 (05) : 1669 - 1680

[3] CD8+ T-CELLS IN PSORIATIC LESIONS PREFERENTIALLY USE T-CELL RECEPTOR V(BETA)3 AND/OR V(BETA)13.1 GENES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (20) : 9282 - 9286

[4] INVOLVEMENT OF HUMAN MUSCLE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNIT GENE (CHRNA) IN SUSCEPTIBILITY TO MYASTHENIA-GRAVIS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (11) : 4668 - 4672

[5] POLYGENIC CONTROL OF AUTOIMMUNE DIABETES IN NONOBESE DIABETIC MICE [J]. NATURE GENETICS, 1993, 4 (04) : 404 - 409

[6] SPONTANEOUS LOSS OF T-CELL TOLERANCE TO GLUTAMIC-ACID DECARBOXYLASE IN MURINE INSULIN-DEPENDENT DIABETES [J]. NATURE, 1993, 366 (6450) : 69 - 72

[7] KENNEDY CG, 1995, NAT GENET, V9, P293

[8] SUPPRESSOR T-CELLS GENERATED BY ORAL TOLERIZATION TO MYELIN BASIC-PROTEIN SUPPRESS BOTH INVITRO AND INVIVO IMMUNE-RESPONSES BY THE RELEASE OF TRANSFORMING GROWTH-FACTOR-BETA AFTER ANTIGEN-SPECIFIC TRIGGERING [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1992, 89 (01) : 421 - 425

[9] OHASHI PS, 1991, CELL, V65, P293

[10] SAKAGUCHI S, 1994, J IMMUNOL, V152, P1471

[1] PREDICTIVE MEDICINE IN AUTOIMMUNE-DISEASES - FROM THE IDENTIFICATION OF GENETIC PREDISPOSITION AND ENVIRONMENTAL INFLUENCE TO PRECOCIOUS IMMUNOTHERAPY [J]. CLINICAL IMMUNOLOGY AND IMMUNOPATHOLOGY, 1994, 72 (02): : 156 - 161

[2] T-CELL-MEDIATED INHIBITION OF THE TRANSFER OF AUTOIMMUNE DIABETES IN NOD MICE [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1989, 169 (05) : 1669 - 1680

[3] CD8+ T-CELLS IN PSORIATIC LESIONS PREFERENTIALLY USE T-CELL RECEPTOR V(BETA)3 AND/OR V(BETA)13.1 GENES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (20) : 9282 - 9286

[4] INVOLVEMENT OF HUMAN MUSCLE ACETYLCHOLINE-RECEPTOR ALPHA-SUBUNIT GENE (CHRNA) IN SUSCEPTIBILITY TO MYASTHENIA-GRAVIS [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (11) : 4668 - 4672

[5] POLYGENIC CONTROL OF AUTOIMMUNE DIABETES IN NONOBESE DIABETIC MICE [J]. NATURE GENETICS, 1993, 4 (04) : 404 - 409

[6] SPONTANEOUS LOSS OF T-CELL TOLERANCE TO GLUTAMIC-ACID DECARBOXYLASE IN MURINE INSULIN-DEPENDENT DIABETES [J]. NATURE, 1993, 366 (6450) : 69 - 72

[7] KENNEDY CG, 1995, NAT GENET, V9, P293

[8] SUPPRESSOR T-CELLS GENERATED BY ORAL TOLERIZATION TO MYELIN BASIC-PROTEIN SUPPRESS BOTH INVITRO AND INVIVO IMMUNE-RESPONSES BY THE RELEASE OF TRANSFORMING GROWTH-FACTOR-BETA AFTER ANTIGEN-SPECIFIC TRIGGERING [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1992, 89 (01) : 421 - 425

[9] OHASHI PS, 1991, CELL, V65, P293

[10] SAKAGUCHI S, 1994, J IMMUNOL, V152, P1471