LEUKOCYTE ADHERENCE-RELATED GLYCOPROTEINS LFA-1, MO1, AND P150,95 - A NEW GROUP OF MONOCLONAL-ANTIBODIES, A NEW DISEASE, AND A POSSIBLE OPPORTUNITY TO UNDERSTAND THE MOLECULAR-BASIS OF LEUKOCYTE ADHERENCE

被引:61
作者
GALLIN, JI
机构
关键词
D O I
10.1093/infdis/152.4.661
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
引用
收藏
页码:661 / 664
页数:4
相关论文
共 35 条
[1]   RECURRENT INFECTIONS AND DELAYED SEPARATION OF THE UMBILICAL-CORD IN AN INFANT WITH ABNORMAL PHAGOCYTIC CELL LOCOMOTION AND OXIDATIVE RESPONSE DURING PARTICLE PHAGOCYTOSIS [J].
ABRAMSON, JS ;
MILLS, EL ;
SAWYER, MK ;
REGELMANN, WR ;
NELSON, JD ;
QUIE, PG .
JOURNAL OF PEDIATRICS, 1981, 99 (06) :887-894
[2]  
AMBRUSO D R, 1982, Pediatric Research, V16, p198A
[3]   ABNORMALITIES OF POLYMORPHONUCLEAR LEUKOCYTE FUNCTION ASSOCIATED WITH A HERITABLE DEFICIENCY OF HIGH MOLECULAR-WEIGHT SURFACE GLYCOPROTEINS (GP138) - COMMON RELATIONSHIP TO DIMINISHED CELL ADHERENCE [J].
ANDERSON, DC ;
SCHMALSTIEG, FC ;
ARNAOUT, MA ;
KOHL, S ;
TOSI, MF ;
DANA, N ;
BUFFONE, GJ ;
HUGHES, BJ ;
BRINKLEY, BR ;
DICKEY, WD ;
ABRAMSON, JS ;
SPRINGER, T ;
BOXER, LA ;
HOLLERS, JM ;
SMITH, CW .
JOURNAL OF CLINICAL INVESTIGATION, 1984, 74 (02) :536-551
[4]   THE SEVERE AND MODERATE PHENOTYPES OF HERITABLE MAC-1, LFA-1 DEFICIENCY - THEIR QUANTITATIVE DEFINITION AND RELATION TO LEUKOCYTE DYSFUNCTION AND CLINICAL-FEATURES [J].
ANDERSON, DC ;
SCHMALSTEIG, FC ;
FINEGOLD, MJ ;
HUGHES, BJ ;
ROTHLEIN, R ;
MILLER, LJ ;
KOHL, S ;
TOSI, MF ;
JACOBS, RL ;
WALDROP, TC ;
GOLDMAN, AS ;
SHEARER, WT ;
SPRINGER, TA .
JOURNAL OF INFECTIOUS DISEASES, 1985, 152 (04) :668-689
[5]   INHIBITION OF PHAGOCYTOSIS OF COMPLEMENT-C3-COATED OR IMMUNOGLOBULIN-G-COATED PARTICLES AND OF C3BI BINDING BY MONOCLONAL-ANTIBODIES TO A MONOCYTE-GRANULOCYTE MEMBRANE GLYCOPROTEIN (MOL) [J].
ARNAOUT, MA ;
TODD, RF ;
DANA, N ;
MELAMED, J ;
SCHLOSSMAN, SF ;
COLTEN, HR .
JOURNAL OF CLINICAL INVESTIGATION, 1983, 72 (01) :171-179
[6]   DEFICIENCY OF A GRANULOCYTE-MEMBRANE GLYCOPROTEIN (GP150) IN A BOY WITH RECURRENT BACTERIAL-INFECTIONS [J].
ARNAOUT, MA ;
PITT, J ;
COHEN, HJ ;
MELAMED, J ;
ROSEN, FS ;
COLTEN, HR .
NEW ENGLAND JOURNAL OF MEDICINE, 1982, 306 (12) :693-699
[7]   ANTI-MAC-1 SELECTIVELY INHIBITS THE MOUSE AND HUMAN TYPE 3 COMPLEMENT RECEPTOR [J].
BELLER, DI ;
SPRINGER, TA ;
SCHREIBER, RD .
JOURNAL OF EXPERIMENTAL MEDICINE, 1982, 156 (04) :1000-1009
[8]   HUMAN-NEUTROPHILS INCREASE EXPRESSION OF C3BI AS WELL AS C3B RECEPTORS UPON ACTIVATION [J].
BERGER, M ;
OSHEA, J ;
CROSS, AS ;
FOLKS, TM ;
CHUSED, TM ;
BROWN, EJ ;
FRANK, MM .
JOURNAL OF CLINICAL INVESTIGATION, 1984, 74 (05) :1566-1571
[9]   SEVERE RECURRENT BACTERIAL-INFECTIONS ASSOCIATED WITH DEFECTIVE ADHERENCE AND CHEMOTAXIS IN 2 PATIENTS WITH NEUTROPHILS DEFICIENT IN A CELL-ASSOCIATED GLYCOPROTEIN [J].
BOWEN, TJ ;
OCHS, HD ;
ALTMAN, LC ;
PRICE, TH ;
VANEPPS, DE ;
BRAUTIGAN, DL ;
ROSIN, RE ;
PERKINS, WD ;
BABIOR, BM ;
KLEBANOFF, SJ ;
WEDGWOOD, RJ .
JOURNAL OF PEDIATRICS, 1982, 101 (06) :932-940
[10]  
BUESCHER ES, 1985, BLOOD, V65