Inflammatory mechanisms in Alzheimer disease: the role of beta-amyloid/glial interactions

被引:0
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作者
J S Pachter
机构
[1] University of Connecticut Health Center,Department of Pharmacology
来源
Molecular Psychiatry | 1997年 / 2卷
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摘要
Recent evidence that insoluble aggregates of beta-amyloid peptide in the brains of Alzheimer patients may elicit neurodestructive immune responses offers the prospect of new therapies to treat this neurodegenerative condition.
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页码:91 / 95
页数:4
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