Identification of variants in genes associated with hypertrophic cardiomyopathy in Mexican patients

被引:0
作者
Catalina García-Vielma
Luis Gerardo Lazalde-Córdova
José Cruz Arzola-Hernández
Erick Noel González-Aceves
Herminio López-Zertuche
Nancy Elena Guzmán-Delgado
Francisco González-Salazar
机构
[1] Instituto Mexicano del Seguro Social,Centro de Investigación Biomédica del Noreste, Departamento de Citogenética
[2] Instituto Mexicano del Seguro Social,Departamento de Electrofisiología
[3] Unidad Médica de Alta Especialidad. Hospital de cardiología No. 34 “Dr. Alfonso J. Treviño Treviño” del Centro Médico Nacional del Noreste,undefined
[4] Instituto Mexicano del Seguro Social,undefined
[5] Hospital General de Zona No. 4,undefined
来源
Molecular Genetics and Genomics | 2023年 / 298卷
关键词
Hypertrophic cardiomyopathy; Sudden death; Gene variants;
D O I
暂无
中图分类号
学科分类号
摘要
The objective of this work was to identify genetic variants in Mexican patients diagnosed with hypertrophic cardiomyopathy (HCM). According to world literature, the genes mainly involved are MHY7 and MYBPC3, although variants have been found in more than 50 genes related to heart disease and sudden death, and to our knowledge there are no studies in the Mexican population. These variants are reported and classified in the ClinVar (PubMed) database and only some of them are recognized in the Online Mendelian Information in Men (OMIM). The present study included 37 patients, with 14 sporadic cases and 6 familial cases, with a total of 21 index cases. Next-generation sequencing was performed on a predesigned panel of 168 genes associated with heart disease and sudden death. The sequencing analysis revealed twelve (57%) pathogenic or probably pathogenic variants, 9 of them were familial cases, managing to identify pathogenic variants in relatives without symptoms of the disease. At the molecular level, nine of the 12 variants (75%) were single nucleotide changes, 2 (17%) deletions, and 1 (8%) splice site alteration. The genes involved were MYH7 (25%), MYBPC3 (25%) and ACADVL, KCNE1, TNNI3, TPM1, SLC22A5, TNNT2 (8%). In conclusion; we found five variants that were not previously reported in public databases. It is important to follow up on the reclassification of variants, especially those of uncertain significance in patients with symptoms of the condition. All patients included in the study and their relatives received family genetic counseling.
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页码:1289 / 1299
页数:10
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