Protective Effects of Inosine on Memory Consolidation in a Rat Model of Scopolamine-Induced Cognitive Impairment: Involvement of Cholinergic Signaling, Redox Status, and Ion Pump Activities

[31] Bild W(1990)Reactive oxygen species formation as a biomarker of methylmercury and trimethyltin neurotoxicity Methods Enzymol 186 407-126

[32] Ababei DC(1972)Glycogen synthase kinase-3 negatively regulates anti-inflammatory interleukin-10 for lipopolysaccharide-induced iNOS/NO biosynthesis and RANTES production in microglial cells J Biol Chem 247 3170-86

[33] Tönnies E(1984)Changes in thiol content and expression of glutathione redox system genes in the hippocampus and cerebellum in Alzheimer's disease Methods Enzymol 105 121-255

[34] Trushina E(2012)Determination of aldehydic lipid peroxidation products: malonaldehyde and 4-hydroxynonenal Eur J Pharmacol 684 79-1350

[35] Amato A(2000)The role of superoxide anion in the autoxidation of epinephrine and a simple assay for superoxide dismutase Dialogues Clin Neurosci 3 247-1828

[36] Terzo S(2010)Catalase in vitro Neurosci Biobehav Rev 34 1307-10

[37] Mulè F(2019)Spermidine decreases Na+, K+-ATPase activity through NMDA receptor and protein kinase G activation in the hippocampus of rats Life Sci 233 116695-794

[38] Moseley A(2017)Pharmacological models of Alzheimer’s disease research Metab Brain Dis 32 1819-339

[39] Williams MT(2019)The validity of scopolamine as a pharmacological model for cognitive impairment: a review of animal behavioral studies Pharmacol Biochem Behav 187 172798-157

[40] Schaefer TL(2004)The cellular and molecular processes associated with scopolamine-induced memory deficit: a model of Alzheimer's biomarkers J Clin Exp Neuropsychol 26 1-1276

[31] Bild W(1990)Reactive oxygen species formation as a biomarker of methylmercury and trimethyltin neurotoxicity Methods Enzymol 186 407-126

[32] Ababei DC(1972)Glycogen synthase kinase-3 negatively regulates anti-inflammatory interleukin-10 for lipopolysaccharide-induced iNOS/NO biosynthesis and RANTES production in microglial cells J Biol Chem 247 3170-86

[33] Tönnies E(1984)Changes in thiol content and expression of glutathione redox system genes in the hippocampus and cerebellum in Alzheimer's disease Methods Enzymol 105 121-255

[34] Trushina E(2012)Determination of aldehydic lipid peroxidation products: malonaldehyde and 4-hydroxynonenal Eur J Pharmacol 684 79-1350

[35] Amato A(2000)The role of superoxide anion in the autoxidation of epinephrine and a simple assay for superoxide dismutase Dialogues Clin Neurosci 3 247-1828

[36] Terzo S(2010)Catalase in vitro Neurosci Biobehav Rev 34 1307-10

[37] Mulè F(2019)Spermidine decreases Na+, K+-ATPase activity through NMDA receptor and protein kinase G activation in the hippocampus of rats Life Sci 233 116695-794

[38] Moseley A(2017)Pharmacological models of Alzheimer’s disease research Metab Brain Dis 32 1819-339

[39] Williams MT(2019)The validity of scopolamine as a pharmacological model for cognitive impairment: a review of animal behavioral studies Pharmacol Biochem Behav 187 172798-157

[40] Schaefer TL(2004)The cellular and molecular processes associated with scopolamine-induced memory deficit: a model of Alzheimer's biomarkers J Clin Exp Neuropsychol 26 1-1276