Epigenome-wide association data implicate DNA methylation as an intermediary of genetic risk in rheumatoid arthritis

被引:0
|
作者
Yun Liu
Martin J Aryee
Leonid Padyukov
M Daniele Fallin
Espen Hesselberg
Arni Runarsson
Lovisa Reinius
Nathalie Acevedo
Margaret Taub
Marcus Ronninger
Klementy Shchetynsky
Annika Scheynius
Juha Kere
Lars Alfredsson
Lars Klareskog
Tomas J Ekström
Andrew P Feinberg
机构
[1] Center for Epigenetics,Department of Medicine
[2] Johns Hopkins University School of Medicine,Department of Oncology
[3] Johns Hopkins University School of Medicine,Department of Medicine
[4] Johns Hopkins University School of Medicine,Department of Biostatistics
[5] Rheumatology Unit,Department of Epidemiology
[6] Karolinska Institute,Department of Biosciences and Nutrition
[7] Center for Molecular Medicine,Department of Medicine Solna
[8] Karolinska Institute,Department of Clinical Neuroscience
[9] Johns Hopkins Bloomberg School of Public Health,undefined
[10] Johns Hopkins Bloomberg School of Public Health,undefined
[11] Karolinska Institute,undefined
[12] Karolinska Institute,undefined
[13] Institute of Environmental Medicine,undefined
[14] Karolinska Institute,undefined
[15] Karolinska Institute,undefined
来源
Nature Biotechnology | 2013年 / 31卷
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学科分类号
摘要
Liu et al. use mediation analysis to find changes in DNA methylation that mediate the genetic risk for rheumatoid arthritis.
引用
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页码:142 / 147
页数:5
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