Microglial-derived microparticles mediate neuroinflammation after traumatic brain injury

[1] Burnier L(2009)Cell-derived microparticles in haemostasis and vascular medicine Thromb Haemost 101 439-451

[2] Fontana P(2016)Microparticles impair hypotensive cerebrovasodilation and cause hippocampal neuronal cell injury after traumatic brain injury J Neurotrauma 33 168-174

[3] Kwak BR(2010)Tissue factor, lipid rafts, and microparticles Semin Thromb Hemost 36 857-864

[4] Angelillo-Scherrer A(2016)Extracellular vesicles and intercellular communication within the nervous system J Clin Invest 126 1198-1207

[5] Bohman LE(2012)Plasmacytoid dendritic cells sequester high prion titres at early stages of prion infection PLoS Pathog 8 e1002538-368

[6] Riley J(2013)Exosome release from infected dendritic cells: a clue for a fast spread of prions in the periphery? J Infect 67 359-265

[7] Milovanova TN(2015)From molecule to molecule and cell to cell: prion-like mechanisms in amyotrophic lateral sclerosis Neurobiol Dis 77 257-15711

[8] Sanborn MR(2013)Mutant copper-zinc superoxide dismutase (SOD1) induces protein secretion pathway alterations and exosome release in astrocytes: implications for disease spreading and motor neuron pathology in amyotrophic lateral sclerosis J Biol Chem 288 15699-11177

[9] Thom SR(2006)Alzheimer’s disease beta-amyloid peptides are released in association with exosomes Proc Natl Acad Sci U S A 103 11172-1800

[10] Armstead WM(2014)Exosome reduction in vivo is associated with lower amyloid plaque load in the 5XFAD mouse model of Alzheimer’s disease Neurobiol Aging 35 1792-1593

[1] Burnier L(2009)Cell-derived microparticles in haemostasis and vascular medicine Thromb Haemost 101 439-451

[2] Fontana P(2016)Microparticles impair hypotensive cerebrovasodilation and cause hippocampal neuronal cell injury after traumatic brain injury J Neurotrauma 33 168-174

[3] Kwak BR(2010)Tissue factor, lipid rafts, and microparticles Semin Thromb Hemost 36 857-864

[4] Angelillo-Scherrer A(2016)Extracellular vesicles and intercellular communication within the nervous system J Clin Invest 126 1198-1207

[5] Bohman LE(2012)Plasmacytoid dendritic cells sequester high prion titres at early stages of prion infection PLoS Pathog 8 e1002538-368

[6] Riley J(2013)Exosome release from infected dendritic cells: a clue for a fast spread of prions in the periphery? J Infect 67 359-265

[7] Milovanova TN(2015)From molecule to molecule and cell to cell: prion-like mechanisms in amyotrophic lateral sclerosis Neurobiol Dis 77 257-15711

[8] Sanborn MR(2013)Mutant copper-zinc superoxide dismutase (SOD1) induces protein secretion pathway alterations and exosome release in astrocytes: implications for disease spreading and motor neuron pathology in amyotrophic lateral sclerosis J Biol Chem 288 15699-11177

[9] Thom SR(2006)Alzheimer’s disease beta-amyloid peptides are released in association with exosomes Proc Natl Acad Sci U S A 103 11172-1800

[10] Armstead WM(2014)Exosome reduction in vivo is associated with lower amyloid plaque load in the 5XFAD mouse model of Alzheimer’s disease Neurobiol Aging 35 1792-1593