Association between furosemide in premature infants and sensorineural hearing loss and nephrocalcinosis: a systematic review

被引:11
作者
Wesley Jackson
Genevieve Taylor
David Selewski
P. Brian Smith
Sue Tolleson-Rinehart
Matthew M. Laughon
机构
[1] University of North Carolina at Chapel Hill,Division of Pediatrics
[2] University of Michigan,Division of Nephrology, Department of Pediatrics and Communicable Diseases, C.S. Mott Children’s Hospital
[3] Duke University Medical Center,Duke Department of Pediatrics
[4] University of North Carolina at Chapel Hill,Gillings School of Global Public Health
关键词
Furosemide; Infant; Premature; Sensorineural hearing loss; Nephrocalcinosis; Nephrolithiasis;
D O I
10.1186/s40748-018-0092-2
中图分类号
学科分类号
摘要
Furosemide is a potent loop diuretic commonly and variably used by neonatologists to improve oxygenation and lung compliance in premature infants. There are several safety concerns with use of furosemide in premature infants, specifically the risk of sensorineural hearing loss (SNHL), and nephrocalcinosis/nephrolithiasis (NC/NL). We conducted a systematic review of all trials and observational studies examining the association between these outcomes with exposure to furosemide in premature infants.
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[1]  
Marshall DD(1999)Risk factors for chronic lung disease in the surfactant era: a North Carolina population-based study of very low birth weight infants. North Carolina Neonatologists Association Pediatrics 104 1345-1350
[2]  
Kotelchuck M(2005)Association between fluid intake and weight loss during the first ten days of life and risk of bronchopulmonary dysplasia in extremely low birth weight infants J Pediatr 147 786-790
[3]  
Young TE(2008)Hearing loss in children with very low birth weight: current review of epidemiology and pathophysiology Arch Dis Child Fetal Neonatal Ed 93 F462-F468
[4]  
Bose CL(2007)Neurological outcomes following preterm birth Semin Fetal Neonatal Med 12 374-382
[5]  
Kruyer L(1971)Ototoxic side-effects of high doses of frusemide in patients with uraemia Postgrad Med J 47 54-56
[6]  
O'Shea TM(1970)Ototoxicity induced by furosemide N Engl J Med 282 1413-1414
[7]  
Oh W(1974)High dose frusemide in acute reversible intrinsic renal failure. A preliminary communication Scott Med J 19 35-39
[8]  
Poindexter BB(1985)Furosemide ototoxicity: clinical and experimental aspects Laryngoscope 95 1-14
[9]  
Perritt R(2004)Na-K-2Cl cotransporter inhibition impairs human lung cellular proliferation Am J Physiol Lung Cell Mol Physiol 287 L510-L514
[10]  
Lemons JA(1986)Ultrasound diagnosis of renal calcification in infants on chronic furosemide therapy J Clin Ultrasound 14 281-287