Outcomes of the 2019 hydrocephalus association workshop, "Driving common pathways: extending insights from posthemorrhagic hydrocephalus"

被引：0
作者：
Jason K. Karimy
Jessie C. Newville
Cameron Sadegh
Jill A. Morris
Edwin S. Monuki
David D. Limbrick
James P. McAllister II
Jenna E. Koschnitzky
Maria K. Lehtinen
Lauren L. Jantzie
机构：
[1] Mountain Area Health Education Center - Boone,Department of Family Medicine
[2] Johns Hopkins Children’s Center,Department of Pediatrics and Neurosurgery
[3] Johns Hopkins School of Medicine,Department of Neurosurgery
[4] Massachusetts General Hospital and Harvard Medical School,National Institute of Neurological Disorders and Stroke, Neuroscience Center
[5] National Institutes of Health,Departments of Pathology & Laboratory Medicine and Developmental & Cell Biology
[6] University of California Irvine,Departments of Neurosurgery and Pediatrics
[7] Washington University School of Medicine in St. Louis,Department of Pathology
[8] Hydrocephalus Association,undefined
[9] Boston Children’s Hospital,undefined
[10] Kennedy Krieger Institute,undefined
来源：
Fluids and Barriers of the CNS | / 20卷
关键词：
Hydrocephalus; Posthemorrhagic; Post-infectious; Intraventricular hemorrhage; Germinal matrix hemorrhage; Cilia; Ependyma; Cerebrospinal fluid;
D O I：
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摘要：
The Hydrocephalus Association (HA) workshop, Driving Common Pathways: Extending Insights from Posthemorrhagic Hydrocephalus, was held on November 4 and 5, 2019 at Washington University in St. Louis. The workshop brought together a diverse group of basic, translational, and clinical scientists conducting research on multiple hydrocephalus etiologies with select outside researchers. The main goals of the workshop were to explore areas of potential overlap between hydrocephalus etiologies and identify drug targets that could positively impact various forms of hydrocephalus. This report details the major themes of the workshop and the research presented on three cell types that are targets for new hydrocephalus interventions: choroid plexus epithelial cells, ventricular ependymal cells, and immune cells (macrophages and microglia).
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