Deletion of newly described pro-survival molecule Pellino-1 increases oxidative stress, downregulates cIAP2/NF-κB cell survival pathway, reduces angiogenic response, and thereby aggravates tissue function in mouse ischemic models

[1] Adluri RS(2011)Disruption of hypoxia-inducible transcription factor-prolyl hydroxylase domain-1 (PHD-1 Antioxid Redox Signal 15 1789-1797

[2] Thirunavukkarasu M(2011)) attenuates ex vivo myocardial ischemia/reperfusion injury through hypoxia-inducible factor-1alpha transcription factor and its target genes in mice J Mol Cell Cardiol 50 239-247

[3] Dunna NR(2010)Thioredoxin 1 enhances neovascularization and reduces ventricular remodeling during chronic myocardial infarction: a study using thioredoxin 1 transgenic mice Antioxid Redox Signal 12 693-702

[4] Zhan L(2011)Preconditioning promotes survival and angiomyogenic potential of mesenchymal stem cells in the infarcted heart via NF-kappaB signaling Lancet 377 1929-1937

[5] Oriowo B(2012)Effect of fibroblast growth factor NV1FGF on amputation and death: a randomised placebo-controlled trial of gene therapy in critical limb ischaemia Eur J Pharmacol 684 116-124

[6] Takeda K(2016)Testosterone replacement therapy promotes angiogenesis after acute myocardial infarction by enhancing expression of cytokines HIF-1a, SDF-1a and VEGF Mod Pathol 29 1313-1323

[7] Sanchez JA(2001)PELI1 expression is correlated with MYC and BCL6 expression and associated with poor prognosis in diffuse large B-cell lymphoma Br J Cancer 84 1322-1329

[8] Otani H(2013)Mutant epidermal growth factor receptor enhances induction of vascular endothelial growth factor by hypoxia and insulin-like growth factor-1 via a PI3 kinase dependent pathway Atherosclerosis 229 396-403

[9] Maulik G(2018)Critical limb ischaemia is characterised by an increased production of whole blood reactive oxygen species and expression of TREM-1 on neutrophils Med Sci Monit 24 8870-8877

[10] Fong GH(2015)Hypoxia-inducible factor 1 alpha (HIF-1alpha)/vascular endothelial growth factor (vegf) pathway participates in angiogenesis of myocardial infarction in muscone-treated mice: preliminary study Semin Cell Dev Biol 39 124-131

[1] Adluri RS(2011)Disruption of hypoxia-inducible transcription factor-prolyl hydroxylase domain-1 (PHD-1 Antioxid Redox Signal 15 1789-1797

[2] Thirunavukkarasu M(2011)) attenuates ex vivo myocardial ischemia/reperfusion injury through hypoxia-inducible factor-1alpha transcription factor and its target genes in mice J Mol Cell Cardiol 50 239-247

[3] Dunna NR(2010)Thioredoxin 1 enhances neovascularization and reduces ventricular remodeling during chronic myocardial infarction: a study using thioredoxin 1 transgenic mice Antioxid Redox Signal 12 693-702

[4] Zhan L(2011)Preconditioning promotes survival and angiomyogenic potential of mesenchymal stem cells in the infarcted heart via NF-kappaB signaling Lancet 377 1929-1937

[5] Oriowo B(2012)Effect of fibroblast growth factor NV1FGF on amputation and death: a randomised placebo-controlled trial of gene therapy in critical limb ischaemia Eur J Pharmacol 684 116-124

[6] Takeda K(2016)Testosterone replacement therapy promotes angiogenesis after acute myocardial infarction by enhancing expression of cytokines HIF-1a, SDF-1a and VEGF Mod Pathol 29 1313-1323

[7] Sanchez JA(2001)PELI1 expression is correlated with MYC and BCL6 expression and associated with poor prognosis in diffuse large B-cell lymphoma Br J Cancer 84 1322-1329

[8] Otani H(2013)Mutant epidermal growth factor receptor enhances induction of vascular endothelial growth factor by hypoxia and insulin-like growth factor-1 via a PI3 kinase dependent pathway Atherosclerosis 229 396-403

[9] Maulik G(2018)Critical limb ischaemia is characterised by an increased production of whole blood reactive oxygen species and expression of TREM-1 on neutrophils Med Sci Monit 24 8870-8877

[10] Fong GH(2015)Hypoxia-inducible factor 1 alpha (HIF-1alpha)/vascular endothelial growth factor (vegf) pathway participates in angiogenesis of myocardial infarction in muscone-treated mice: preliminary study Semin Cell Dev Biol 39 124-131