Ion channels boost axonal injury in multiple sclerosis

被引:0
作者
Reinhard Hohlfeld
机构
[1] Reinhard Hohlfeld is in the Institute of Clinical Neuroimmunology,
[2] Ludwig Maximilians University of Munich,undefined
[3] and is a member of the Munich Cluster for Systems Neurology (SyNergy),undefined
[4] Munich,undefined
[5] Germany.,undefined
来源
Nature Medicine | 2012年 / 18卷
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摘要
In neuroinflammatory diseases such as multiple sclerosis, ion channels may fan the embers of neurodegeneration. A new study shows that the cation channel TRPM4 (transient receptor potential melastatin 4) crucially contributes to axonal loss in an animal model of multiple sclerosis (pages 1805–1811).
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页码:1743 / 1745
页数:2
相关论文
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