ILC2 regulates hyperoxia-induced lung injury via an enhanced Th17 cell response in the BPD mouse model

被引:0
作者
Yue Zhu
Lanlan Mi
Hongyan Lu
Huimin Ju
Xiaobo Hao
Suqing Xu
机构
[1] The Affiliated Hospital of Jiangsu University,Department of Pediatrics
[2] Shanghai Children’s Medical Center,Department of Neonatology
来源
BMC Pulmonary Medicine | / 23卷
关键词
Bronchopulmonary dysplasia; Lung injury; Th17 cell response; Type 2 innate lymphoid cells;
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[1]  
Thébaud B(2019)Bronchopulmonary dysplasia Nat Rev Dis Primers 5 78-38
[2]  
Goss KN(2018)Recent advances in Bronchopulmonary Dysplasia: pathophysiology, Prevention, and treatment Lung 196 129-68
[3]  
Laughon M(1967)Pulmonary disease following respirator therapy of hyaline-membrane disease. Bronchopulmonary dysplasia N Engl J Med 276 357-37
[4]  
Whitsett JA(2020)Ventilation strategies in severe bronchopulmonary dysplasia Neoreviews 21 e226-93
[5]  
Abman SH(2021)Hyperoxia-induced bronchopulmonary dysplasia: better models for better therapies Dis Model Mech 14 047753-18
[6]  
Steinhorn RH(2006)Hyperoxia causes angiopoietin 2-mediated acute lung injury and necrotic cell death Nat Med 12 1286-64
[7]  
Aschner JL(2015)The NLRP3 inflammasome is critically involved in the development of bronchopulmonary dysplasia Nat Commun 6 8977-40
[8]  
Davis PG(2021)Cytokine-regulated Th17 plasticity in human health and diseases Immunology 163 3-9
[9]  
McGrath-Morrow SA(2019)Inflammasome activation and Th17 responses Mol Immunol 107 142-508
[10]  
Soll RF(2014)Maternal inflammation modulates infant immune response patterns to viral lung challenge in a murine model Pediatr Res 76 33-52