Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes

被引：0

作者：

Omar Lozano

Christian Silva-Platas

Héctor Chapoy-Villanueva

Baruc E. Pérez

Jarmon G. Lees

Chrishan J. A. Ramachandra

Flavio F. Contreras-Torres

Anay Lázaro-Alfaro

Estefanía Luna-Figueroa

Judith Bernal-Ramírez

Aldemar Gordillo-Galeano

Alfredo Benitez

Yuriana Oropeza-Almazán

Elena C. Castillo

Poh Ling Koh

Derek J. Hausenloy

Shiang Y. Lim

Gerardo García-Rivas

机构：

[1] Cátedra de Cardiología y Medicina Vascular,Tecnologico de Monterrey. Escuela Nacional de Medicina y Ciencias de la Salud

[2] Tecnologico de Monterrey. Centro de Investigación Biomédica,Departments of Medicine and Surgery

[3] Hospital Zambrano-Helión,O’Brien Institute Department

[4] University of Melbourne,National Heart Research Institute Singapore

[5] St Vincent’s Institute of Medical Research,Cardiovascular and Metabolic Disorders Programme

[6] National Heart Centre Singapore,Tecnologico de Monterrey

[7] Duke-NUS Medical School,Department of Physics and Astronomy

[8] Escuela de Ingeniería y Ciencias,Yong Loo Lin School of Medicine

[9] The University of Texas at San Antonio,The Hatter Cardiovascular Institute

[10] National University Singapore,Cardiovascular Research Center

[11] University College London,undefined

[12] College of Medical and Health Sciences,undefined

[13] Asia University,undefined

来源：

Particle and Fibre Toxicology | / 17卷

关键词：

Mitochondria; Silica nanoparticles; Oxidative stress; Cardiotoxicity; Permeability transition; Heart; Calcium overload;

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