Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytes

被引:0
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作者
Omar Lozano
Christian Silva-Platas
Héctor Chapoy-Villanueva
Baruc E. Pérez
Jarmon G. Lees
Chrishan J. A. Ramachandra
Flavio F. Contreras-Torres
Anay Lázaro-Alfaro
Estefanía Luna-Figueroa
Judith Bernal-Ramírez
Aldemar Gordillo-Galeano
Alfredo Benitez
Yuriana Oropeza-Almazán
Elena C. Castillo
Poh Ling Koh
Derek J. Hausenloy
Shiang Y. Lim
Gerardo García-Rivas
机构
[1] Cátedra de Cardiología y Medicina Vascular,Tecnologico de Monterrey. Escuela Nacional de Medicina y Ciencias de la Salud
[2] Tecnologico de Monterrey. Centro de Investigación Biomédica,Departments of Medicine and Surgery
[3] Hospital Zambrano-Helión,O’Brien Institute Department
[4] University of Melbourne,National Heart Research Institute Singapore
[5] St Vincent’s Institute of Medical Research,Cardiovascular and Metabolic Disorders Programme
[6] National Heart Centre Singapore,Tecnologico de Monterrey
[7] Duke-NUS Medical School,Department of Physics and Astronomy
[8] Escuela de Ingeniería y Ciencias,Yong Loo Lin School of Medicine
[9] The University of Texas at San Antonio,The Hatter Cardiovascular Institute
[10] National University Singapore,Cardiovascular Research Center
[11] University College London,undefined
[12] College of Medical and Health Sciences,undefined
[13] Asia University,undefined
来源
Particle and Fibre Toxicology | / 17卷
关键词
Mitochondria; Silica nanoparticles; Oxidative stress; Cardiotoxicity; Permeability transition; Heart; Calcium overload;
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