Determinants of the postprandial triglyceride response to a high-fat meal in healthy overweight and obese adults

[1] Burton BT(1985)Health implications of obesity: an NIH Consensus Development Conference Int J Obes. 9 155-70

[2] Foster WR(2001)Postprandial dyslipidemia in men with visceral obesity: an effect of reduced LDL receptor expression? American Journal of Physiology-Endocrinology Metabolism 281 E626-E32-81

[3] Hirsch J(2001)Metabolic and Lifestyle Determinants of Postprandial Lipemia Differ From Those of Fasting Triglycerides Arteriosclerosis, Thrombosis, and Vascular Biology. 21 275-333

[4] Van Itallie TB(2011)Mechanisms, Pathophysiology, and Management of Obesity Triglycerides Cardiovascular Disease Circulation 123 2292-66

[5] Mamo JCL(2017)Nonfasting versus fasting lipid profile for cardiovascular risk prediction N Engl J Med 376 254-41

[6] Watts GF(2019)Fasting and nonfasting lipid levels: influence of normal food intake on lipids, lipoproteins, apolipoproteins, and cardiovascular risk prediction Pathology 51 131-56

[7] Barrett PHR(2008)Postprandial lipemia and cardiovascular disease risk: Interrelationships between dietary, physiological and genetic determinants Circulation 118 2047-33

[8] Smith D(2012)Fasting compared with nonfasting triglycerides and risk of cardiovascular events in women Atherosclerosis 220 22-16

[9] James AP(2007)Impaired glucose tolerance is a risk factor for cardiovascular disease, but not impaired fasting glucose. The Funagata Diabetes Study Jama 298 309-4

[10] Pal S(1999)Nonfasting triglycerides and risk of myocardial infarction, ischemic heart disease, and death in men and women Diabetes Care 22 920-308

[1] Burton BT(1985)Health implications of obesity: an NIH Consensus Development Conference Int J Obes. 9 155-70

[2] Foster WR(2001)Postprandial dyslipidemia in men with visceral obesity: an effect of reduced LDL receptor expression? American Journal of Physiology-Endocrinology Metabolism 281 E626-E32-81

[3] Hirsch J(2001)Metabolic and Lifestyle Determinants of Postprandial Lipemia Differ From Those of Fasting Triglycerides Arteriosclerosis, Thrombosis, and Vascular Biology. 21 275-333

[4] Van Itallie TB(2011)Mechanisms, Pathophysiology, and Management of Obesity Triglycerides Cardiovascular Disease Circulation 123 2292-66

[5] Mamo JCL(2017)Nonfasting versus fasting lipid profile for cardiovascular risk prediction N Engl J Med 376 254-41

[6] Watts GF(2019)Fasting and nonfasting lipid levels: influence of normal food intake on lipids, lipoproteins, apolipoproteins, and cardiovascular risk prediction Pathology 51 131-56

[7] Barrett PHR(2008)Postprandial lipemia and cardiovascular disease risk: Interrelationships between dietary, physiological and genetic determinants Circulation 118 2047-33

[8] Smith D(2012)Fasting compared with nonfasting triglycerides and risk of cardiovascular events in women Atherosclerosis 220 22-16

[9] James AP(2007)Impaired glucose tolerance is a risk factor for cardiovascular disease, but not impaired fasting glucose. The Funagata Diabetes Study Jama 298 309-4

[10] Pal S(1999)Nonfasting triglycerides and risk of myocardial infarction, ischemic heart disease, and death in men and women Diabetes Care 22 920-308