Sex difference in the interrelationship between TNF-α and oxidative stress status in first-episode drug-naïve schizophrenia

[1] van Os J(2008)Gene-environment interactions in schizophrenia: review of epidemiological findings and future directions Schizophr Bull 34 1066-1082

[2] Rutten BP(2020)Lowered antioxidant defenses and increased oxidative toxicity are hallmarks of deficit schizophrenia: a nomothetic network psychiatry approach Mol Neurobiol 57 4578-4597

[3] Poulton R(2020)Maternal immune activation causes schizophrenia-like behaviors in the offspring through activation of immune-inflammatory, oxidative and apoptotic pathways, and lowered antioxidant defenses and neuroprotection Mol Neurobiol 57 4345-4361

[4] Maes M(2020)Cytokines, oxidative stress and cellular markers of inflammation in schizophrenia Curr Top Behav Neurosci 44 49-66

[5] Sirivichayakul S(2018)Meta-analysis of cerebrospinal fluid cytokine and tryptophan catabolite alterations in psychiatric patients: comparisons between schizophrenia, bipolar disorder, and depression Schizophr Bull 44 75-83

[6] Matsumoto AK(2018)Cytokine alterations in first-episode schizophrenia and bipolar disorder: relationships to brain structure and symptoms J Neuroinflammation 15 165-6

[7] Michelin AP(2019)JNK signalling mediates aspects of maternal immune activation: importance of maternal genotype in relation to schizophrenia risk J Neuroinflammation 16 18-284

[8] de Oliveira SL(2018)Interaction between BDNF and TNF-alpha genes in schizophrenia Psychoneuroendocrinology 89 1-1709

[9] de Lima Pedrao JV(2018)TNF-alpha and IL-6 are associated with the deficit syndrome and negative symptoms in patients with chronic schizophrenia Schizophr Res 199 281-890

[10] Moreira EG(2016)A meta-analysis of blood cytokine network alterations in psychiatric patients: comparisons between schizophrenia, bipolar disorder and depression Mol Psychiatry 21 1696-2

[1] van Os J(2008)Gene-environment interactions in schizophrenia: review of epidemiological findings and future directions Schizophr Bull 34 1066-1082

[2] Rutten BP(2020)Lowered antioxidant defenses and increased oxidative toxicity are hallmarks of deficit schizophrenia: a nomothetic network psychiatry approach Mol Neurobiol 57 4578-4597

[3] Poulton R(2020)Maternal immune activation causes schizophrenia-like behaviors in the offspring through activation of immune-inflammatory, oxidative and apoptotic pathways, and lowered antioxidant defenses and neuroprotection Mol Neurobiol 57 4345-4361

[4] Maes M(2020)Cytokines, oxidative stress and cellular markers of inflammation in schizophrenia Curr Top Behav Neurosci 44 49-66

[5] Sirivichayakul S(2018)Meta-analysis of cerebrospinal fluid cytokine and tryptophan catabolite alterations in psychiatric patients: comparisons between schizophrenia, bipolar disorder, and depression Schizophr Bull 44 75-83

[6] Matsumoto AK(2018)Cytokine alterations in first-episode schizophrenia and bipolar disorder: relationships to brain structure and symptoms J Neuroinflammation 15 165-6

[7] Michelin AP(2019)JNK signalling mediates aspects of maternal immune activation: importance of maternal genotype in relation to schizophrenia risk J Neuroinflammation 16 18-284

[8] de Oliveira SL(2018)Interaction between BDNF and TNF-alpha genes in schizophrenia Psychoneuroendocrinology 89 1-1709

[9] de Lima Pedrao JV(2018)TNF-alpha and IL-6 are associated with the deficit syndrome and negative symptoms in patients with chronic schizophrenia Schizophr Res 199 281-890

[10] Moreira EG(2016)A meta-analysis of blood cytokine network alterations in psychiatric patients: comparisons between schizophrenia, bipolar disorder and depression Mol Psychiatry 21 1696-2