Effects of early recombinant human growth hormone treatment in young Chinese children with Prader–Willi syndrome

被引:0
作者
Ying Gao
Li-Li Yang
Yang-Li Dai
Zheng Shen
Qiong Zhou
Chao-Chun Zou
机构
[1] The Children’s Hospital of Zhejiang University School of Medicine,
[2] Hangzhou children’s Hospital,undefined
来源
Orphanet Journal of Rare Diseases | / 18卷
关键词
Prader–Willi syndrome; Growth hormone; Body composition; Length; Height; Carbohydrate metabolism; Spinal deformity;
D O I
暂无
中图分类号
学科分类号
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[1]  
Cassidy SB(2012)Prader–Willi syndrome Genet Med Offl J Am College Med Genet 14 10-26
[2]  
Schwartz S(2001)Genome organization, function, and imprinting in Prader–Willi and Angelman syndromes Annu Rev Genomics Hum Genet 2 153-175
[3]  
Miller JL(2004)Minimum prevalence, birth incidence and cause of death for Prader–Willi syndrome in Flanders Eur J Hum Genet 12 238-240
[4]  
Driscoll DJ(2019)A retrospective analysis of the prevalence of imprinting disorders in Estonia from 1998 to 2016 Eur J Hum Genet 27 1649-1658
[5]  
Nicholls RD(2009)A deletion of the HBII-85 class of small nucleolar RNAs (snoRNAs) is associated with hyperphagia, obesity and hypogonadism Hum Mol Genet 18 3257-3265
[6]  
Knepper JL(2019)Genotype-phenotype relationships and endocrine findings in Prader–Willi syndrome Front Endocrinol 10 864-255
[7]  
Vogels A(2015)Highly restricted deletion of the SNORD116 region is implicated in Prader–Willi syndrome Eur J Hum Genet 23 252-1201
[8]  
Van Den Ende J(2010)Paternally inherited microdeletion at 15q11.2 confirms a significant role for the SNORD116 C/D box snoRNA cluster in Prader-Willi syndrome Eur J Hum Gen 18 1196-721
[9]  
Keymolen K(2008)Prader–Willi phenotype caused by paternal deficiency for the HBII-85 C/D box small nucleolar RNA cluster Nat Genet 40 719-443
[10]  
Mortier G(2017)SNORD116 deletions cause Prader–Willi syndrome with a mild phenotype and macrocephaly Clin Genet 92 440-376