The transcription repressors Bach2 and Bach1 promote B cell development by repressing the myeloid program

被引：0

作者：

Ari Itoh-Nakadai

Reina Hikota

Akihiko Muto

Kohei Kometani

Miki Watanabe-Matsui

Yuki Sato

Masahiro Kobayashi

Atsushi Nakamura

Yuichi Miura

Yoko Yano

Satoshi Tashiro

Jiying Sun

Tomokatsu Ikawa

Kyoko Ochiai

Tomohiro Kurosaki

Kazuhiko Igarashi

机构：

[1] Tohoku University Graduate School of Medicine,Department of Biochemistry

[2] CREST,Department of Cellular Biology

[3] Japan Science and Technology Agency,undefined

[4] RIKEN Center for Integrative Medical Sciences,undefined

[5] Research Institute for Radiation Biology and Medicine,undefined

[6] Graduate School of Biomedical Science,undefined

[7] Hiroshima University,undefined

[8] Laboratory for Immune Regeneration RIKEN Center for Integrative Medical Sciences,undefined

[9] Center for Regulatory Epigenome and Diseases,undefined

[10] Tohoku University Graduate School of Medicine,undefined

[11] WPI Immunology Frontier Research Center,undefined

[12] Osaka University,undefined

来源：

Nature Immunology | 2014年 / 15卷

关键词：

D O I：

暂无

中图分类号：

学科分类号：

摘要：

The role of the repressors Bach1 and Bach2 in early B cell development is unclear. Igarashi and colleagues have now found these Bach factors directly repress various myeloid genes in common lymphoid progenitors to restrict a B cell lineage fate.

引用

页码：1171 / 1180

页数：9

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