Autoregulation: mediators and renin–angiotensin system in diseases and treatments

[1] Vaughan CJ(2000)Hypertensive emergencies The Lancet 356 411-417

[2] Delanty N(2008)Theoretical model of blood flow autoregulation: roles of myogenic, shear-dependent, and metabolic responses Am J Physiol Heart Circulat Physiol 295 H1572-H1579

[3] Carlson BE(1987)Autoregulation of the coronary circulation Prog Cardiovasc Dis 29 293-323

[4] Arciero JC(2011)Modulation of the myogenic response in renal blood flow autoregulation by NO depends on endothelial nitric oxide synthase (eNOS), but not neuronal or inducible NOS J Physiol 589 4731-4744

[5] Secomb TW(2008)Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin Br J Pharmacol 154 1225-1235

[6] Dole WP(2007)Angiotensin-(1–7) through receptor Mas mediates endothelial nitric oxide synthase activation via Akt-dependent pathways Hypertension 49 185-192

[7] Dautzenberg M(2001)Mixed messages: modulation of inflammation and immune responses by prostaglandins and thromboxanes J Clin Investig 108 15-23

[8] Keilhoff G(2002)Non-steroidal anti-inflammatory drugs and the continuum of renal dysfunction J Hyperten 20 S17-S23

[9] Just A(2004)TXAS-deleted mice exhibit normal thrombopoiesis, defective hemostasis, and resistance to arachidonate-induced death Blood 104 135-142

[10] Xavier FE(2021)Comparative study on the effect of aspirin, TP receptor antagonist and TxA2 synthase inhibitor on the vascular tone of human saphenous vein and internal mammary artery Life Sci 286 1762-1767

[1] Vaughan CJ(2000)Hypertensive emergencies The Lancet 356 411-417

[2] Delanty N(2008)Theoretical model of blood flow autoregulation: roles of myogenic, shear-dependent, and metabolic responses Am J Physiol Heart Circulat Physiol 295 H1572-H1579

[3] Carlson BE(1987)Autoregulation of the coronary circulation Prog Cardiovasc Dis 29 293-323

[4] Arciero JC(2011)Modulation of the myogenic response in renal blood flow autoregulation by NO depends on endothelial nitric oxide synthase (eNOS), but not neuronal or inducible NOS J Physiol 589 4731-4744

[5] Secomb TW(2008)Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin Br J Pharmacol 154 1225-1235

[6] Dole WP(2007)Angiotensin-(1–7) through receptor Mas mediates endothelial nitric oxide synthase activation via Akt-dependent pathways Hypertension 49 185-192

[7] Dautzenberg M(2001)Mixed messages: modulation of inflammation and immune responses by prostaglandins and thromboxanes J Clin Investig 108 15-23

[8] Keilhoff G(2002)Non-steroidal anti-inflammatory drugs and the continuum of renal dysfunction J Hyperten 20 S17-S23

[9] Just A(2004)TXAS-deleted mice exhibit normal thrombopoiesis, defective hemostasis, and resistance to arachidonate-induced death Blood 104 135-142

[10] Xavier FE(2021)Comparative study on the effect of aspirin, TP receptor antagonist and TxA2 synthase inhibitor on the vascular tone of human saphenous vein and internal mammary artery Life Sci 286 1762-1767