Inflammation-driven bone formation in a mouse model of ankylosing spondylitis: sequential not parallel processes

被引:0
作者
Hsu-Wen Tseng
Miranda E. Pitt
Tibor T. Glant
Allan F. McRae
Tony J. Kenna
Matthew A. Brown
Allison R. Pettit
Gethin P. Thomas
机构
[1] The University of Queensland Diamantina Institute,Section of Molecular Medicine, Department of Orthopedic Surgery
[2] Translational Research Institute,undefined
[3] Rush University Medical Center,undefined
[4] The University of Queensland,undefined
[5] Queensland Brain Institute,undefined
[6] The University of Queensland-Mater Research Institute,undefined
[7] Translational Research Institute,undefined
来源
Arthritis Research & Therapy | / 18卷
关键词
Ankylosing spondylitis; Proteoglycan-induced spondylitis mouse model; Enthesis; Intervertebral disc destruction; Chondroidal bone formation; Spondyloarthropathy; Arthritis; Osteoproliferation;
D O I
暂无
中图分类号
学科分类号
摘要
引用
收藏
相关论文
共 202 条
[1]  
Benjamin M(2009)Evidence for a distinctive pattern of bone formation in enthesophytes Ann Rheum Dis 68 1003-10
[2]  
Toumi H(2000)Histopathologic evidence that sacroiliitis in ankylosing spondylitis is not merely enthesitis Arthritis Rheum 43 2011-24
[3]  
Suzuki D(2011)Analysis of IL-17+ cells in facet joints of patients with spondyloarthritis suggests that the innate immune pathway might be of greater relevance than the Th17-mediated adaptive immune response Arthritis Res Ther 13 R95-51
[4]  
Hayashi K(2006)Immunohistologic analysis of zygapophyseal joints in patients with ankylosing spondylitis Arthritis Rheum 54 2845-13
[5]  
McGonagle D(2006)Immunohistochemical analysis of hip arthritis in ankylosing spondylitis: evaluation of the bone-cartilage interface and subchondral bone marrow Arthritis Rheum 54 1805-9
[6]  
Francois RJ(2013)In situ analysis of interleukin-23- and interleukin-12-positive cells in the spine of patients with ankylosing spondylitis Arthritis Rheum 65 1522-7
[7]  
Gardner DL(2005)A longitudinal study on an autoimmune murine model of ankylosing spondylitis Ann Rheum Dis 64 981-52
[8]  
Degrave EJ(2012)Excessive bone formation in a mouse model of ankylosing spondylitis is associated with decreases in Wnt pathway inhibitors Arthritis Res Ther 14 R253-5
[9]  
Bywaters EG(2002)IL-4 and IL-12 regulate proteoglycan-induced arthritis through Stat-dependent mechanisms J Immunol 169 3345-9
[10]  
Appel H(1988)The relationship between pelvic, spinal and hip involvement in ankylosing spondylitis--one disease process or several? Br J Rheumatol 27 393-20