Indomethacin-responsive headache syndromes

被引：67
作者：
Dodick D.W. [1 ]
机构：
[1] Department of Neurology, Mayo Medical School, Mayo Clinic, Scottsdale, AZ 85259
来源：
Current Pain and Headache Reports | 2004年 / 8卷 / 1期
关键词：
Indomethacin; Cluster Headache; Primary Headache Disorder; Paroxysmal Hemicrania; Hemicrania Continuum;
D O I：
10.1007/s11916-004-0036-6
中图分类号：
学科分类号：
摘要：
Indomethacin-responsive headache syndromes represent a unique group of primary headache disorders characterized by a prompt and often complete response to indomethacin to the exclusion of other nonsteroidal anti-inflammatory drugs and medications usually effective in treating other primary headache disorders. Because these headache disorders can easily be overlooked in clinical practice, they likely are more common than previously recognized. Indomethacin-responsive headache syndromes can be divided into several distinct categories: a select group of trigeminal-autonomic cephalgias, valsalva-induced headaches, and primary stabbing headache (ice-pick headache or jabs and jolts syndrome). Each category can be differentiated clinically and by the extent to which the individual headache disorders respond to indomethacin. The paroxysmal and continuous hemicranias invariably respond in an absolute manner to indomethacin, whereas valsalva-induced and ice-pick headaches may respond in an equally dramatic, but somewhat less consistent fashion. Hypnic headache recently has been described as another primary headache disorder that may respond to indomethacin. Copyright © 2004 by Current Science Inc.
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页码：19 / 26
页数：7
相关论文
共 63 条
[1]  
Goadsby P.J., Lipton R.B., A review of paroxysmal hemicranias, SUNCT syndrome and other short-lasting headaches with autonomic features, including new cases, Brain, 120, pp. 193-209, (1997)
[2]  
Goadsby P.J., Edvinsson L., The trigeminovascular system and migraine: Studies characterizing cerebrovascular and neuropeptide changes seen in humans and cats, Ann. Neurol., 33, pp. 48-56, (1993)
[3]  
Goadsby P.J., Edvinsson L., Human in vivo evidence for trigeminovascular activation in cluster headache: Neuropeptide changes and effects of acute attacks therapies, Brain, 117, pp. 427-434, (1994)
[4]  
Goadsby P.J., Edvinsson L., Neuropeptide changes in a case of chronic paroxysmal hemicrania: Evidence for trigemino-parasympathetic activation, Cephalalgia, 16, pp. 448-450, (1996)
[5]  
May A., Bahra A., Buchel Q., Et al., Hypothalamic activation in cluster headache attacks, Lancet, 352, pp. 275-278, (1998)
[6]  
May A., Bahra A., Buchel C., Et al., Functional magnetic resonance imaging in spontaneous attacks of SUNCT: Short-lasting neuralgiform headache with conjunctival injection and tearing, Ann. Neurol., 46, pp. 787-790, (1999)
[7]  
Pascual J., Iglesias F., Oterino A., Et al., Cough, exertional, and sexual headaches: An analysis of 72 benign and symptomatic cases, Neurology, 46, pp. 1520-1524, (1996)
[8]  
Raskin N.H., The cough headache syndrome: Treatment, Neurology, 45, (1995)
[9]  
Antonacci F., Sjaastad O., Chronic paroxysmal hemicrania (CPH): A review of the clinical manifestations, Headache, 29, pp. 648-656, (1989)
[10]  
Boes C., Dodick D.W., The clinical spectrum of chronic paroxysmal hemicrania seen at the Mayo Clinic from 1976-1996, Headache, (2003)
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