TRIM22 suppresses Zika virus replication by targeting NS1 and NS3 for proteasomal degradation

被引：0
作者：
Shulong Zu
Chunfeng Li
Lili Li
Yong-Qiang Deng
Xiang Chen
Dan Luo
Qing Ye
Yi-Jiao Huang
Xiao-Feng Li
Rong-Rong Zhang
Nina Sun
Xianqi Zhang
Saba R. Aliyari
Karin Nielsen-Saines
Jae U. Jung
Heng Yang
Cheng-Feng Qin
Genhong Cheng
机构：
[1] Chinese Academy of Medical Sciences and Peking Union Medical College,Center for Systems Medicine, Institute of Basic Medical Sciences
[2] Chinese Academy of Medical Sciences and Peking Union Medical College,Institute of Systems Medicine
[3] Suzhou Institute of Systems Medicine,State Key Laboratory of Pathogen and Biosecurity, Beijing Institute of Microbiology and Epidemiology
[4] Academy of Military Medical Sciences,Institute for Immunity, Transplantation and Infection, Department of Pathology, Department of Microbiology and Immunology
[5] Stanford University,Department of Microbiology, Immunology and Molecular Genetics
[6] University of California,Division of Pediatric Infectious Diseases, David Geffen School of Medicine
[7] University of California,Department of Cancer Biology and Global Center for Pathogens Research and Human Health
[8] Lerner Research Institute,undefined
[9] Cleveland Clinic,undefined
来源：
Cell & Bioscience | / 12卷
关键词：
TRIM22; Zika virus; Infection; Nonstructural protein; Ubiquitination;
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