The M50I polymorphic substitution in association with the R263K mutation in HIV-1 subtype B integrase increases drug resistance but does not restore viral replicative fitness

被引:0
作者
Melissa Wares
Thibault Mesplède
Peter K Quashie
Nathan Osman
Yingshan Han
Mark A Wainberg
机构
[1] McGill University AIDS Centre,Lady Davis Institute for Medical Research
[2] Jewish General Hospital,Department of Microbiology and Immunology, Faculty of Medicine
[3] McGill University,Division of Experimental Medicine, Faculty of Medicine
[4] McGill University,undefined
来源
Retrovirology | / 11卷
关键词
HIV integrase; Subtype B; Antiretrovirals; R263K; Resistance mutation; M50I; Polymorphism; INSTI-naïve;
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