Liraglutide dictates macrophage phenotype in apolipoprotein E null mice during early atherosclerosis

被引:0
|
作者
Robyn Bruen
Sean Curley
Sarina Kajani
Daniel Crean
Marcella E. O’Reilly
Margaret B. Lucitt
Catherine G. Godson
Fiona C. McGillicuddy
Orina Belton
机构
[1] UCD Conway Institute,Diabetes Complications Research Centre, School of Biomolecular and Biomedical Science
[2] University College Dublin,Diabetes Complications Research Centre, School of Medicine
[3] UCD Conway Institute,Diabetes Complications Research Centre, School of Veterinary Medicine
[4] University College Dublin,School of Medicine, Department of Pharmacology and Therapeutics
[5] UCD Conway Institute,undefined
[6] University College Dublin,undefined
[7] Trinity College Dublin,undefined
[8] The University of Dublin,undefined
来源
关键词
MΦ2 macrophages; Monocytes; Anti-inflammatory; Plaque microenvironment; Atherosclerosis;
D O I
暂无
中图分类号
学科分类号
摘要
引用
收藏
相关论文
共 50 条
  • [21] Atherosclerosis development in apolipoprotein E-null mice deficient for CD69
    Gomez, Manuel
    Sanz-Gonzalez, Silvia M.
    Abu Nabah, Yafa Naim
    Lamana, Amalia
    Sanchez-Madrid, Francisco
    Andres, Vicente
    CARDIOVASCULAR RESEARCH, 2009, 81 (01) : 197 - 205
  • [22] APOLIPOPROTEIN-E PHENOTYPE, HYPERLIPIDEMIA AND ATHEROSCLEROSIS
    DAVIGNON, J
    ROEDERER, G
    UNION MEDICALE DU CANADA, 1988, 117 (01): : 56 - 61
  • [23] Glucose-Dependent Insulinotropic Polypeptide Prevents the Progression of Macrophage-Driven Atherosclerosis in Diabetic Apolipoprotein E-Null Mice
    Nogi, Yukinori
    Nagashima, Masaharu
    Terasaki, Michishige
    Nohtomi, Kyoko
    Watanabe, Takuya
    Hirano, Tsutomu
    PLOS ONE, 2012, 7 (04):
  • [24] Glucose-dependent insulinotropic polypeptide prevents the progression of macrophage-driven atherosclerosis in diabetic apolipoprotein E-null mice
    Masaharu, N.
    Nogi, Y.
    Terasaki, M.
    Notomi, K.
    Tomoyasu, M.
    Hirano, T.
    DIABETOLOGIA, 2012, 55 : S323 - S324
  • [25] Aspirin but not Meloxicam Attenuates Early Atherosclerosis in Apolipoprotein E Knockout Mice
    Kraus, Sarah
    Naumov, Inna
    Shapira, Shiran
    Kazanov, Dina
    Aroch, Ilan
    Afek, Arnon
    Eisenberg, Oded
    George, Jacob
    Arber, Nadir
    Finkelstein, Ariel
    ISRAEL MEDICAL ASSOCIATION JOURNAL, 2014, 16 (04): : 233 - 238
  • [26] Macrophage phenotype in mice deficient in both macrophage-colony-stimulating factor (Op) and apolipoprotein E
    de Villiers, WJS
    Smith, JD
    Miyata, M
    Dansky, HM
    Darley, E
    Gordon, S
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 1998, 18 (04) : 631 - 640
  • [27] Propagermanium reduces atherosclerosis in apolipoprotein E knockout mice via inhibition of macrophage infiltration
    Yamashita, T
    Kawashima, S
    Ozaki, M
    Namiki, M
    Inoue, N
    Hirata, K
    Yokoyama, M
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2002, 22 (06) : 969 - 974
  • [28] Inhibition of monocyte/macrophage infiltration by propagermanium reduces atherosclerosis in apolipoprotein E knockout mice
    Yamashita, T
    Kawashima, S
    Ozaki, M
    Takeuchi, S
    Inoue, N
    Hirata, KI
    CIRCULATION, 2000, 102 (18) : 308 - 308
  • [29] Deficiency of Glycine N-Methyltransferase Aggravates Atherosclerosis in Apolipoprotein E-Null Mice
    Chien-Yu Chen
    Li-Chieh Ching
    Yi-Jen Liao
    Yuan-Bin Yu
    Chia-Yuan Tsou
    Song-Kun Shyue
    Yi-Ming Arthur Chen
    Tzong-Shyuan Lee
    Molecular Medicine, 2012, 18 : 744 - 752
  • [30] Null mutation of peroxiredoxin II ameliorates atherosclerosis in apolipoprotein E knock-out mice
    Park, JG
    Choi, JH
    Lee, YM
    Lee, MR
    Lee, MN
    Yoo, JY
    Kim, CG
    Cho, CS
    Kim, YM
    Rhee, SG
    Yu, DY
    Oh, GT
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2006, 26 (05) : E98 - E98