Molecular mechanism of immune response, synovial proliferation and apoptosis in rheumatoid arthritis

被引:0
作者
T. Hasunuma
T. Kato
T. Kobata
K. Nishioka
机构
[1] St. Marianna University School of Medicine,Rheumatology, Immunology and Genetics Program, Institute of Medical Science
来源
Springer Seminars in Immunopathology | 1998年 / 20卷
关键词
Rheumatoid Arthritis; Immune Response; Arthritis; Internal Medicine; Molecular Mechanism;
D O I
暂无
中图分类号
学科分类号
摘要
引用
收藏
页码:41 / 52
页数:11
相关论文
共 430 条
  • [81] Emmrich F(1995)Apoptosis in rheumatoid arthritis synovium J Clin Invest 96 1631-undefined
  • [82] Kroczek RA(1996)B cells in systemic autoimmune disease: recent insights from Fas-deficient mice and men Curr Opin Immunol 8 852-undefined
  • [83] Hasunuma T(1996)Apoptosis mediated by the Fas system Prog Mol Subcell Biol 16 87-undefined
  • [84] Hoa TTM(1996)Greatly accelerated lymphadenopathy and autoimmune disease in Ipr mice lacking tumor necrosis factor receptor 1 J Immunol 156 1661-undefined
  • [85] Aono H(1995)Fas and Fas ligand: Ipr and gld mutations Immunol Today 16 39-undefined
  • [86] Asahara H(1996)Constitutive expression of bel-2 in B cells causes a lethal form of lupus like autoimmune disease after induction of neonatal tolerance to H-2b alloantigens J Exp Med 183 2523-undefined
  • [87] Yonehara S(1996)Inhibition of apoptosis and augmentation of lymphoproliferation in bcl-2 transgenic Fas/Fas ligand-defective mice Cell Immunol 168 220-undefined
  • [88] Yamamoto K(1994)Autoimmune disease: a problem of defective apoptosis Arthritis Rheum 317 1415-undefined
  • [89] Sumida T(1994)The bcl-2 proto-oncogene is overexpressed in systemic lupus erythematosus J Autoimmun 7 623-undefined
  • [90] Gay S(1994)High levels of bcl-2 protein in circulating T lymphocytes, but not B lymphocytes, of patients with systemic lupus erythematosus Arthritis Rheum 37 1423-undefined