Processing the H3K36me3 signature

被引：0
|
作者：
Robert J Sims III
Danny Reinberg
机构：
[1] Robert J. Sims III is at Constellation Pharmaceuticals,Danny Reinberg is at the Department of Biochemistry
[2] Cambridge,undefined
[3] Massachusetts 02139,undefined
[4] USA.,undefined
[5] New York University School of Medicine,undefined
[6] New York 10016 USA. reinbd01@nyumc.org robert.sims@constellationpharma.com,undefined
来源：
Nature Genetics | 2009年 / 41卷
关键词：
D O I：
暂无
中图分类号：
学科分类号：
摘要：
The global patterning of histone lysine methylation has been scrutinized over the years in an effort to uncover unique features indicative of chromatin function. A study in Caenorhabditis elegans now shows that nucleosomes covering exons and introns on active genes are differentially marked by H3K36 trimethylation, suggesting a new mode of communication between chromatin and pre-mRNA processing.
引用
收藏
页码：270 / 271
页数：1
相关论文
共 50 条
  • [31] Targeted Profiling of Epitranscriptomic Reader, Writer, and Eraser Proteins Regulated by H3K36me3
    Yin, Jiekai
    Qi, Tianyu F.
    Li, Lin
    Wang, Yinsheng
    ANALYTICAL CHEMISTRY, 2023, 95 (25) : 9672 - 9679
  • [32] Regulation of SETD2 stability is important for the fidelity of H3K36me3 deposition
    Bhattacharya, Saikat
    Workman, Jerry L.
    EPIGENETICS & CHROMATIN, 2020, 13 (01)
  • [33] Molecular basis for H3K36me3 recognition by the Tudor domain of PHF1
    Musselman, Catherine A.
    Avvakumov, Nikita
    Watanabe, Reiko
    Abraham, Christopher G.
    Lalonde, Marie-Eve
    Hong, Zehui
    Allen, Christopher
    Roy, Siddhartha
    Nunez, James K.
    Nickoloff, Jac
    Kulesza, Caroline A.
    Yasui, Akira
    Cote, Jacques
    Kutateladze, Tatiana G.
    NATURE STRUCTURAL & MOLECULAR BIOLOGY, 2012, 19 (12) : 1266 - +
  • [34] Regulation of SETD2 stability is important for the fidelity of H3K36me3 deposition
    Saikat Bhattacharya
    Jerry L. Workman
    Epigenetics & Chromatin, 13
  • [35] H3K9me3, H3K36me3, and H4K20me3 Expression Correlates with Patient Outcome in Esophageal Squamous Cell Carcinoma as Epigenetic Markers
    Menghan Zhou
    Yiping Li
    Shaofeng Lin
    Yanping Chen
    Yanyan Qian
    Zhujiang Zhao
    Hong Fan
    Digestive Diseases and Sciences, 2019, 64 : 2147 - 2157
  • [36] Modifications of H3K9me2, H3K36me3 and H4K20me2 may be involved in arsenic-induced genetic damage
    Li, Jun
    Ma, Lu
    Wang, Xilan
    Li, Daochuan
    Zeng, Qibing
    Xing, Xiumei
    Li, Chenggui
    Xie, Lang
    Chen, Li
    Chen, Wen
    Zhang, Aihua
    TOXICOLOGY RESEARCH, 2016, 5 (05) : 1380 - 1387
  • [37] SETD2-mediated crosstalk between H3K36me3 and H3K79me2 in MLL-rearranged leukemia
    J Bu
    A Chen
    X Yan
    F He
    Y Dong
    Y Zhou
    J He
    D Zhan
    P Lin
    Y Hayashi
    Y Sun
    Y Zhang
    Z Xiao
    H L Grimes
    Q F Wang
    G Huang
    Leukemia, 2018, 32 : 890 - 899
  • [38] A novel synthetic lethal interaction between the histone mark H3K36me3 and checkpoint kinases
    Pfister, S. X.
    Markkanen, E.
    Jiang, Y.
    Sarkar, S.
    D'Angiolella, V.
    Dianov, G.
    Ryan, A. J.
    Humphrey, T. C.
    EUROPEAN JOURNAL OF CANCER, 2014, 50 : 71 - 71
  • [39] SETD2-mediated crosstalk between H3K36me3 and H3K79me2 in MLL-rearranged leukemia
    Bu, J.
    Chen, A.
    Yan, X.
    He, F.
    Dong, Y.
    Zhou, Y.
    He, J.
    Zhan, D.
    Lin, P.
    Hayashi, Y.
    Sun, Y.
    Zhang, Y.
    Xiao, Z.
    Grimes, H. L.
    Wang, Q. F.
    Huang, G.
    LEUKEMIA, 2018, 32 (04) : 890 - 899
  • [40] Erratum: H3K36me3 key to Polycomb-mediated gene silencing in lineage specification
    Jumana AlHaj Abed
    Richard S. Jones
    Nature Structural & Molecular Biology, 2013, 20 : 244 - 244
12345