Dengue virus sero-cross-reactivity drives antibody-dependent enhancement of infection with zika virus

被引:0
|
作者
Wanwisa Dejnirattisai
Piyada Supasa
Wiyada Wongwiwat
Alexander Rouvinski
Giovanna Barba-Spaeth
Thaneeya Duangchinda
Anavaj Sakuntabhai
Van-Mai Cao-Lormeau
Prida Malasit
Felix A Rey
Juthathip Mongkolsapaya
Gavin R Screaton
机构
[1] Hammersmith Campus,Division of Immunology and Inflammation, Department of Medicine
[2] Imperial College London,Department of Immunology
[3] Dengue Hemorrhagic Fever Research Unit,Département de Virologie
[4] Office for Research and Development,undefined
[5] Siriraj Hospital,undefined
[6] Faculty of Medicine,undefined
[7] Mahidol University,undefined
[8] Graduate Program in Immunology,undefined
[9] Faculty of Medicine,undefined
[10] Siriraj Hospital,undefined
[11] Mahidol University,undefined
[12] Institut Pasteur,undefined
[13] Unité de Virologie Structurale,undefined
[14] CNRS UMR 3569 Virologie,undefined
[15] Medical Biotechnology Unit,undefined
[16] National Center for Genetic Engineering and Biotechnology,undefined
[17] National Science and Technology Development Agency,undefined
[18] Institut Pasteur,undefined
[19] Functional Genetics of Infectious Diseases Unit,undefined
[20] CNRS URA3012,undefined
[21] Unit of Emerging Infectious Diseases,undefined
[22] Institut Louis Malardé,undefined
来源
Nature Immunology | 2016年 / 17卷
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摘要
Infection with Zika virus has occurred in areas previously exposed to dengue virus, a closely related flavivirus. Screaton and colleagues find that monoclonal antibodies to dengue virus cross-react with Zika virus and enhance infection.
引用
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页码:1102 / 1108
页数:6
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