Hyperexcitability of neurons in field Ca1 evoked by transient episodes of hypoxia in hippocampal slices from rats of different ages

[1] Kalemenev S.V., Savin A.V., Levin S.G., Godukhin S.G., Long-term potentiation of glutamatergic transmission and epileptiform activity evoked by transient episodes of anoxia in field CA1 of rat hippocampal slices, Ros. Fiziol. Zh. Im. I. M. Sechenova, 86, 12, pp. 1676-1681, (2000)

[2] Alger B.E., Nicoll R.A., Feed-forward dendritic inhibition in rat hippocampal pyramidal cells studied in vitro, J. Physiol. (London), 328, pp. 105-123, (1982)

[3] Avoli M., Synaptic activation of GABA A receptors causes a depolarizing potential under physiological conditions in rat hippocampal pyramidal cells, Eur. J. Neurosci., 4, pp. 416-426, (1992)

[4] Bergamasco B., Benna P., Ferrero P., Gavinelli R., Neonatal hypoxia and epileptic risk: A clinical perspective study, Epilepsia, 25, 2, pp. 131-136, (1984)

[5] Crain B.J., Westerkam W.D., Harrison A.H., Nadler J.V., Selective neuronal death after transient forebrain ischemia in the Mongolian gerbil: A silver impregnation study, J. Neurosci., 10, pp. 387-402, (1988)

[6] Godukhin O., Savin A., Kalemenev S., Levin S., Neuronal hyperexcitability induced by repeated brief episodes of hypoxia in rat hippocampal slices: Involvement of ionotropic glutamate receptors and L-type Ca²⁺ channels, Neuropharmacol., 42, pp. 459-466, (2002)

[7] Luhmann H.J., Ischemia and lesion induced imbalances in cortical function, Progr. Neurobiol., 48, pp. 131-166, (1996)

[8] Jensen F.E., Wang C., Hypoxia induces hyperexcitability in vivo and in vitro in the immature hippocampus, Epilepsy Res., 26, 1, pp. 131-140, (1996)

[9] Jensen F.E., Wang C., Stafstrom C.E., Liu Z., Geary C., Stevens M.C., Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia in vivo, J. Neurophysiol., 79, pp. 73-81, (1998)

[10] Semyanov A., Godukhin O., Kindling-like state in rat hippocampal CA1 slices induced by the repeated short-term extracellular K⁺ increases: The role of L-type Ca²⁺ channels, Neurosci. Lett., 223, pp. 177-180, (1997)

[1] Kalemenev S.V., Savin A.V., Levin S.G., Godukhin S.G., Long-term potentiation of glutamatergic transmission and epileptiform activity evoked by transient episodes of anoxia in field CA1 of rat hippocampal slices, Ros. Fiziol. Zh. Im. I. M. Sechenova, 86, 12, pp. 1676-1681, (2000)

[2] Alger B.E., Nicoll R.A., Feed-forward dendritic inhibition in rat hippocampal pyramidal cells studied in vitro, J. Physiol. (London), 328, pp. 105-123, (1982)

[3] Avoli M., Synaptic activation of GABA A receptors causes a depolarizing potential under physiological conditions in rat hippocampal pyramidal cells, Eur. J. Neurosci., 4, pp. 416-426, (1992)

[4] Bergamasco B., Benna P., Ferrero P., Gavinelli R., Neonatal hypoxia and epileptic risk: A clinical perspective study, Epilepsia, 25, 2, pp. 131-136, (1984)

[5] Crain B.J., Westerkam W.D., Harrison A.H., Nadler J.V., Selective neuronal death after transient forebrain ischemia in the Mongolian gerbil: A silver impregnation study, J. Neurosci., 10, pp. 387-402, (1988)

[6] Godukhin O., Savin A., Kalemenev S., Levin S., Neuronal hyperexcitability induced by repeated brief episodes of hypoxia in rat hippocampal slices: Involvement of ionotropic glutamate receptors and L-type Ca²⁺ channels, Neuropharmacol., 42, pp. 459-466, (2002)

[7] Luhmann H.J., Ischemia and lesion induced imbalances in cortical function, Progr. Neurobiol., 48, pp. 131-166, (1996)

[8] Jensen F.E., Wang C., Hypoxia induces hyperexcitability in vivo and in vitro in the immature hippocampus, Epilepsy Res., 26, 1, pp. 131-140, (1996)

[9] Jensen F.E., Wang C., Stafstrom C.E., Liu Z., Geary C., Stevens M.C., Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia in vivo, J. Neurophysiol., 79, pp. 73-81, (1998)

[10] Semyanov A., Godukhin O., Kindling-like state in rat hippocampal CA1 slices induced by the repeated short-term extracellular K⁺ increases: The role of L-type Ca²⁺ channels, Neurosci. Lett., 223, pp. 177-180, (1997)