共 226 条
[1]
Graham DI(1992)Diffuse axonal injury caused by assault J Clin Pathol. 45 840-841
[2]
Clark JC(1996)Altered β-APP metabolism after head injury and its relationship to the aetiology of Alzheimer’s disease Acta Neurochir Suppl. 66 96-102
[3]
Adams JH(2012)Understanding secondary injury Q Rev Biol. 87 89-127
[4]
Gennarelli TA(2006)Extending the understanding of sensory neuropeptides Eur J Pharmacol. 533 171-181
[5]
Graham DI(2004)Neurogenic inflammation is associated with development of edema and functional deficits following traumatic brain injury in rats Neuropeptides. 38 40-47
[6]
Gentleman SM(2010)Substance P immunoreactivity increases following human traumatic brain injury Acta Neurochir Suppl. 106 211-216
[7]
Nicoll JA(1988)Neurokinin A in cerebral vessels: characterization, localization and effects in vitro Regul Pept. 20 181-197
[8]
Royston MC(1995)An investigation into the mechanism of capsaicin-induced oedema in rabbit skin Br J Pharmacol. 114 570-577
[9]
McKenzie JE(2009)Substance P is associated with the development of brain edema and functional deficits after traumatic brain injury J Cereb Blood Flow Metab. 29 1388-1398
[10]
Roberts GW(2011)A substance P antagonist reduces axonal injury and improves neurologic outcome when administered up to 12 hours after traumatic brain injury J Neurotrauma. 28 217-224