Control of cytokine mRNA degradation by the histone deacetylase inhibitor ITF2357 in rheumatoid arthritis fibroblast-like synoviocytes: beyond transcriptional regulation

被引：0
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作者：
Chiara Angiolilli
Pawel A. Kabala
Aleksander M. Grabiec
Marzia Rossato
Wi S. Lai
Gianluca Fossati
Paolo Mascagni
Christian Steinkühler
Perry J. Blackshear
Kris A. Reedquist
Dominique L. Baeten
Timothy R. D. J. Radstake
机构：
[1] University Medical Center Utrecht,Laboratory of Translational Immunology and Department of Rheumatology and Clinical Immunology
[2] Academic Medical Center/University of Amsterdam,Amsterdam Rheumatology and Immunology Center, Department of Clinical Immunology and Rheumatology and Department of Experimental Immunology
[3] Jagiellonian University,Department of Microbiology, Faculty of Biochemistry, Biophysics and Biotechnology
[4] University of Verona,Functional Genomics Center
[5] National Institute of Environmental Health Sciences,Signal Transduction Laboratory
[6] Italfarmaco Research and Development,undefined
来源：
Arthritis Research & Therapy | / 20卷
关键词：
Rheumatoid arthritis; Fibroblast-like synoviocytes; Inflammation; mRNA stability; Tristetraprolin; Histone deacetylase inhibitor; ITF2357;
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