Comprehensive methylation analysis of imprinting-associated differentially methylated regions in colorectal cancer

[1] Tomizawa S(2012)Genomic imprinting and its relevance to congenital disease, infertility, molar pregnancy and induced pluripotent stem cell J Hum Genet 57 84-91

[2] Sasaki H(2013)Epigenetic and genetic alterations of the imprinting disorder Beckwith-Wiedemann syndrome and related disorders J Hum Genet 58 402-409

[3] Soejima H(2011)The role of imprinted genes in fetal growth abnormalities Birth Defects Res A Clin Mol Teratol 91 682-692

[4] Higashimoto K(2008)Different mechanisms cause imprinting defects at the IGF2/H19 locus in Beckwith-Wiedemann syndrome and Wilms’ tumour Hum Mol Genet 17 1427-1435

[5] Piedrahita JA(2012)Mechanisms of activation of the paternally expressed genes by the Prader-Willi imprinting center in the Prader-Willi/Angelman syndromes domains Proc Natl Acad Sci U S A 109 7403-7408

[6] Cerrato F(2006)Genomic imprinting and cancer: from primordial germ cells to somatic cells ScientificWorldJournal 6 1888-1910

[7] Sparago A(2007)Epigenetic specificity of loss of imprinting of the IGF2 gene in Wilms tumors J Natl Cancer Inst 99 1270-1273

[8] Verde G(2008)Loss of imprinting of IGF2 correlates with hypermethylation of the H19 differentially methylated region in hepatoblastoma Br J Cancer 99 1891-1899

[9] De Crescenzo A(2004)Silencing of imprinted CDKN1C gene expression is associated with loss of CpG and histone H3 lysine 9 methylation at DMR-LIT1 in esophageal cancer Oncogene 23 4380-4388

[10] Citro V(2008)Imprinted tumor suppressor genes ARHI and PEG3 are the most frequently down-regulated in human ovarian cancers by loss of heterozygosity and promoter methylation Cancer 112 1489-1502

[1] Tomizawa S(2012)Genomic imprinting and its relevance to congenital disease, infertility, molar pregnancy and induced pluripotent stem cell J Hum Genet 57 84-91

[2] Sasaki H(2013)Epigenetic and genetic alterations of the imprinting disorder Beckwith-Wiedemann syndrome and related disorders J Hum Genet 58 402-409

[3] Soejima H(2011)The role of imprinted genes in fetal growth abnormalities Birth Defects Res A Clin Mol Teratol 91 682-692

[4] Higashimoto K(2008)Different mechanisms cause imprinting defects at the IGF2/H19 locus in Beckwith-Wiedemann syndrome and Wilms’ tumour Hum Mol Genet 17 1427-1435

[5] Piedrahita JA(2012)Mechanisms of activation of the paternally expressed genes by the Prader-Willi imprinting center in the Prader-Willi/Angelman syndromes domains Proc Natl Acad Sci U S A 109 7403-7408

[6] Cerrato F(2006)Genomic imprinting and cancer: from primordial germ cells to somatic cells ScientificWorldJournal 6 1888-1910

[7] Sparago A(2007)Epigenetic specificity of loss of imprinting of the IGF2 gene in Wilms tumors J Natl Cancer Inst 99 1270-1273

[8] Verde G(2008)Loss of imprinting of IGF2 correlates with hypermethylation of the H19 differentially methylated region in hepatoblastoma Br J Cancer 99 1891-1899

[9] De Crescenzo A(2004)Silencing of imprinted CDKN1C gene expression is associated with loss of CpG and histone H3 lysine 9 methylation at DMR-LIT1 in esophageal cancer Oncogene 23 4380-4388

[10] Citro V(2008)Imprinted tumor suppressor genes ARHI and PEG3 are the most frequently down-regulated in human ovarian cancers by loss of heterozygosity and promoter methylation Cancer 112 1489-1502