Clinical value of plasmablasts in predicting disease relapse in patients with IgG4-related disease

被引:0
作者
Yiwen Wang
Zheng Zhao
Dai Gao
Hui Wang
Simin Liao
Gui Luo
Xiaojian Ji
Yan Li
Xiuru Wang
Yurong Zhao
Kunpeng Li
Jie Zhang
Jingyu Jin
Yamei Zhang
Jian Zhu
Jianglin Zhang
Feng Huang
机构
[1] Chinese PLA General Hospital,Department of Rheumatology and Immunology, First Medical Center
来源
Clinical Rheumatology | 2023年 / 42卷
关键词
Biomarker; Disease relapse; Glucocorticoids; IgG4-related disease; Immunosuppressive agents; Leflunomide; Plasmablasts;
D O I
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学科分类号
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页码:135 / 143
页数:8
相关论文
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[1]  
Pillai S(2020)Immune mechanisms of fibrosis and inflammation in IgG4-related disease Curr Opin Rheumatol 32 146-151
[2]  
Perugino C(2020)IgG4-related disease: an update on pathophysiology and implications for clinical care Nat Rev Rheumatol 16 702-714
[3]  
Kaneko N(2018)A CD8alpha- subset of CD4+SLAMF7+ cytotoxic t cells is expanded in patients with igg4-related disease and decreases following glucocorticoid treatment Arthritis Rheum 70 1133-1143
[4]  
Perugino CA(2015)Number of circulating follicular helper 2 t cells correlates with IgG4 and interleukin-4 levels and plasmablast numbers in IgG4-related disease Arthritis Rheumatol 67 2476-2481
[5]  
Stone JH(2014)De novo oligoclonal expansions of circulating plasmablasts in active and relapsing IgG4-related disease J Allergy Clin Immunol 134 679-687
[6]  
Della-Torre E(2014)Circulating Th2 memory cells in IgG4-related disease are restricted to a defined subset of subjects with atopy Allergy 69 399-402
[7]  
Bozzalla-Cassione E(2017)Clonally expanded cytotoxic CD4+ T cells and the pathogenesis of IgG4-related disease Autoimmunity 50 19-24
[8]  
Sciorati C(2015)Plasmablasts as a biomarker for IgG4-related disease, independent of serum IgG4 concentrations Ann Rheum Dis 74 190-195
[9]  
Ruggiero E(2016)Clonal expansion of CD4(+) cytotoxic T lymphocytes in patients with IgG4-related disease J Allergy Clin Immunol 138 825-838
[10]  
Lanzillotta M(2020)B lymphocytes directly contribute to tissue fibrosis in patients with IgG(4)-related disease J Allergy Clin Immunol 145 968-981.e914