Integrative profiling of Epstein–Barr virus transcriptome using a multiplatform approach

[11] Rickinson A(2003)Synergistic autoactivation of the Epstein–Barr virus immediate-early BRLF1 promoter by Rta and Zta Virology 310 199-18

[12] de Martel C(2019)BZLF1 interacts with chromatin remodelers promoting escape from latent infections with EBV Life Sci Alliance 2 1-2850

[13] Georges D(2011)Initiation of Epstein–Barr virus Lytic replication requires transcription and the formation of a stable RNA-DNA hybrid molecule at orilyt J Virol 85 2837-24638

[14] Bray F(2019)Four-dimensional analyses show that replication compartments are clonal factories in which Epstein–Barr viral DNA amplification is coordinated Proc Natl Acad Sci USA 116 24630-2265

[15] Ferlay J(2019)An epigenetic journey: Epstein–Barr virus transcribes chromatinized and subsequently unchromatinized templates during its lytic cycle J Virol 93 2247-26

[16] Clifford GM(2018)CAGE-seq analysis of Epstein–Barr virus lytic gene transcription: 3 kinetic classes from 2 mechanisms PLoS Pathog 14 1-265

[17] Gold JE(2013)Epstein–Barr virus and host cell methylation: regulation of latency, replication and virus reactivation Curr Opin Virol 3 260-2565

[18] Okyay RA(2006)Virus and cell RNAs Expressed during Epstein–Barr Virus replication J Virol 80 2548-245

[19] Licht WE(2011)Coupled transcriptome and proteome analysis of human lymphotropic tumor viruses: Insights on the detection and discovery of viral genes BMC Genom 12 233-1493

[20] Hurley DJ(2012)An atlas of the Epstein–Barr virus transcriptome and epigenome reveals host-virus regulatory interactions Cell Host Microbe 19 1479-1616

[11] Rickinson A(2003)Synergistic autoactivation of the Epstein–Barr virus immediate-early BRLF1 promoter by Rta and Zta Virology 310 199-18

[12] de Martel C(2019)BZLF1 interacts with chromatin remodelers promoting escape from latent infections with EBV Life Sci Alliance 2 1-2850

[13] Georges D(2011)Initiation of Epstein–Barr virus Lytic replication requires transcription and the formation of a stable RNA-DNA hybrid molecule at orilyt J Virol 85 2837-24638

[14] Bray F(2019)Four-dimensional analyses show that replication compartments are clonal factories in which Epstein–Barr viral DNA amplification is coordinated Proc Natl Acad Sci USA 116 24630-2265

[15] Ferlay J(2019)An epigenetic journey: Epstein–Barr virus transcribes chromatinized and subsequently unchromatinized templates during its lytic cycle J Virol 93 2247-26

[16] Clifford GM(2018)CAGE-seq analysis of Epstein–Barr virus lytic gene transcription: 3 kinetic classes from 2 mechanisms PLoS Pathog 14 1-265

[17] Gold JE(2013)Epstein–Barr virus and host cell methylation: regulation of latency, replication and virus reactivation Curr Opin Virol 3 260-2565

[18] Okyay RA(2006)Virus and cell RNAs Expressed during Epstein–Barr Virus replication J Virol 80 2548-245

[19] Licht WE(2011)Coupled transcriptome and proteome analysis of human lymphotropic tumor viruses: Insights on the detection and discovery of viral genes BMC Genom 12 233-1493

[20] Hurley DJ(2012)An atlas of the Epstein–Barr virus transcriptome and epigenome reveals host-virus regulatory interactions Cell Host Microbe 19 1479-1616