Tollip interaction with STAT3: a novel mechanism to regulate human airway epithelial responses to type 2 cytokines

被引:0
作者
Niccolette Schaunaman
Kris Genelyn Dimasuay
Monica Kraft
Hong Wei Chu
机构
[1] National Jewish Health,Department of Medicine
[2] University of Arizona,Department of Medicine
[3] National Jewish Health,undefined
[4] College of Medicine,undefined
[5] Tucson,undefined
来源
Respiratory Research | / 23卷
关键词
Tollip; STAT3; Airway epithelial cells; IL-13; Type 2 inflammation; Protein–protein interaction;
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[1]  
Kowalski EJA(2017)Toll-interacting protein in resolving and non-resolving inflammation Front Immunol 8 511-1390
[2]  
Li L(2019)TOLLIP deficiency is associated with increased resistance to Mucosal Immunol 12 1382-355
[3]  
Shah JA(2013) pneumonia Genes Immun 14 347-1746
[4]  
Emery R(2007)Toll-like receptor-associated sequence variants and prostate cancer risk among men of African descent BMC Dermatol 7 3-1482
[5]  
Lee B(2012)Association of toll-interacting protein gene polymorphisms with atopic dermatitis J Immunol 189 1737-210
[6]  
Venkatasubramanian S(2015)Human TOLLIP regulates TLR2 and TLR4 signaling and its polymorphisms are associated with susceptibility to tuberculosis Am J Respir Crit Care Med 192 1475-1563
[7]  
Simmons JD(2015)TOLLIP, MUC5B, and the response to N-acetylcysteine among individuals with idiopathic pulmonary fibrosis PLoS Negl Trop Dis 9 e0003875-233
[8]  
Brown M(2017)Polymorphisms in the TOLLIP gene influence susceptibility to cutaneous Leishmaniasis caused by Brain Behav Immun 59 200-395
[9]  
Rogers EN(2016) in the Amazonas state of Brazil Clin Exp Allergy 46 1549-65
[10]  
Jones DZ(2019)Toll-interacting protein deficiency promotes neurodegeneration via impeding autophagy completion in high-fat diet-fed ApoE(-/-) mouse model Clin Rev Allergy Immunol 56 219-175