Porphyromonas gingivalis and the pathogenesis of rheumatoid arthritis: analysis of various compartments including the synovial tissue

[1] MacGregor AJ(2000)Characterizing the quantitative genetic contribution to rheumatoid arthritis using data from twins Arthritis Rheum 43 30-37

[2] Snieder H(2003)Cutting edge: the conversion of arginine to citrulline allows for a high-affinity peptide interaction with the rheumatoid arthritis-associated HLA-DRB1 *0401 MHC class II molecule J Immunol 171 538-541

[3] Rigby AS(2004)A gene-environment interaction between smoking and shared epitope genes in HLA-DR provides a high risk of seropositive rheumatoid arthritis Arthritis Rheum 50 3085-3092

[4] Koskenvuo M(2003)The conversion of arginine to citrulline allows for a high-affinity peptide interaction with the rheumatoid arthritis associated HLA-DRB1*0401 MHC class II molecule J Immunol 171 538-541

[5] Kaprio J(1987)Molecular mimicry and autoimmune disease Cell 50 819-820

[6] Aho K(1998)Detection of bacterial DNA in joint samples from patients with undifferentiated arthritis and reactive arthritis, using polymerase chain reaction with universal 16S ribosomal RNA primers Arthritis Rheum 41 535-543

[7] Silman AJ(2000)Presence of bacterial DNA and bacterial peptidoglycans in joints of patients with rheumatoid arthritis and other arthritides Arthritis Rheum 43 593-598

[8] Hill JA(1998)Life below the gum line: pathogenic mechanisms of Microbiol Mol Biol Rev 62 1244-1263

[9] Southwood S(2004)Hypothesis: the humoral immune response to oral bacteria provides a stimulus for the development of rheumatoid arthritis Inflammation 28 311-318

[10] Sette A(2011)Rheumatoid arthritis and periodontitis; a possible link via citrullination Anaerobe 17 196-200

[1] MacGregor AJ(2000)Characterizing the quantitative genetic contribution to rheumatoid arthritis using data from twins Arthritis Rheum 43 30-37

[2] Snieder H(2003)Cutting edge: the conversion of arginine to citrulline allows for a high-affinity peptide interaction with the rheumatoid arthritis-associated HLA-DRB1 *0401 MHC class II molecule J Immunol 171 538-541

[3] Rigby AS(2004)A gene-environment interaction between smoking and shared epitope genes in HLA-DR provides a high risk of seropositive rheumatoid arthritis Arthritis Rheum 50 3085-3092

[4] Koskenvuo M(2003)The conversion of arginine to citrulline allows for a high-affinity peptide interaction with the rheumatoid arthritis associated HLA-DRB1*0401 MHC class II molecule J Immunol 171 538-541

[5] Kaprio J(1987)Molecular mimicry and autoimmune disease Cell 50 819-820

[6] Aho K(1998)Detection of bacterial DNA in joint samples from patients with undifferentiated arthritis and reactive arthritis, using polymerase chain reaction with universal 16S ribosomal RNA primers Arthritis Rheum 41 535-543

[7] Silman AJ(2000)Presence of bacterial DNA and bacterial peptidoglycans in joints of patients with rheumatoid arthritis and other arthritides Arthritis Rheum 43 593-598

[8] Hill JA(1998)Life below the gum line: pathogenic mechanisms of Microbiol Mol Biol Rev 62 1244-1263

[9] Southwood S(2004)Hypothesis: the humoral immune response to oral bacteria provides a stimulus for the development of rheumatoid arthritis Inflammation 28 311-318

[10] Sette A(2011)Rheumatoid arthritis and periodontitis; a possible link via citrullination Anaerobe 17 196-200