The receptors CD96 and CD226 oppose each other in the regulation of natural killer cell functions

被引：0

作者：

Christopher J Chan

Ludovic Martinet

Susan Gilfillan

Fernando Souza-Fonseca-Guimaraes

Melvyn T Chow

Liam Town

David S Ritchie

Marco Colonna

Daniel M Andrews

Mark J Smyth

机构：

[1] Cellular Immunity Laboratory,Department of Immunology

[2] Cancer Immunology Program,Department of Pathology and Immunology

[3] Trescowthick Research Laboratories,Sir Peter MacCallum Department of Oncology

[4] Peter MacCallum Cancer Centre,undefined

[5] Monash University,undefined

[6] Alfred Medical Research and Education Precinct,undefined

[7] Immunology in Cancer and Infection Laboratory,undefined

[8] QIMR Berghofer Medical Research Institute,undefined

[9] Washington University School of Medicine,undefined

[10] Hematology Immunology Translational Research Laboratory,undefined

[11] Peter MacCallum Cancer Centre,undefined

[12] University of Melbourne,undefined

[13] School of Medicine,undefined

[14] University of Queensland,undefined

来源：

Nature Immunology | 2014年 / 15卷

关键词：

D O I：

暂无

中图分类号：

学科分类号：

摘要：

The function of the lymphocyte-expressed receptor CD96 is almost entirely unknown. Smyth and colleagues demonstrate that it serves a key role in restraining activation of NK cells in part by competing with the activating receptor CD226 (DNAM-1).

引用

页码：431 / 438

页数：7

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