New agents, new ideas and new hope

被引：0

作者：

Titte R. Srinivas

Bruce Kaplan

机构：

[1] Glickman Urological and Kidney Institute,Department of Nephrology and Hypertension

[2] University of Arizona School of Medicine,Division of Nephrology

来源：

Nature Reviews Nephrology | 2012年 / 8卷

关键词：

D O I：

暂无

中图分类号：

学科分类号：

摘要：

The past year was marked by several excellent studies that represent important therapeutic advances in kidney transplantation or that further our understanding of the genetic basis of chronic allograft dysfunction, clinical tolerance and outcomes of kidney transplantation.

引用

页码：74 / 75

页数：1

共 15 条

[1]

Larsen CP(2005)Rational development of LEA29Y (belatacept), a high-affinity variant of CTLA4-Ig with potent immunosuppressive properties Am. J. Transplant. 5 443-453

[2]

Vincenti F(2010)A phase III study of belatacept-based immunosuppression regimens versus cyclosporine in renal transplant recipients (BENEFIT study) Am. J. Transplant. 10 535-546

[3]

Montgomery RA(2011)Desensitization in HLA-incompatible kidney recipients and survival N. Engl. J. Med. 365 318-326

[4]

Friedman DJ(2011)Population-based risk assessment of APOL1 on renal disease J. Am. Soc. Nephrol. 22 2098-2105

[5]

Kozlitina J(2011)The Am. J. Transplant. 11 1025-1030

[6]

Genovese G(2010) gene and allograft survival after kidney transplantation J. Clin. Invest. 120 1836-1847

[7]

Jog P(2011)Identification of a B cell signature associated with renal transplant tolerance in humans Am. J. Transplant. 11 1916-1926

[8]

Pollak MR(2005)Comparison of transcriptional and blood cell-phenotypic markers between operationally tolerant liver and kidney recipients JAMA 294 2726-2733

[9]

Reeves-Daniel AM(2011)Deceased-donor characteristics and the survival benefit of kidney transplantation Am. J. Transplant. 11 2657-2664

[10]

Newell KA(undefined)Influence of CIT-induced DGF on kidney transplant outcomes undefined undefined undefined-undefined

← 1 2 →