Salidroside protects endothelial cells against LPS-induced inflammatory injury by inhibiting NLRP3 and enhancing autophagy

被引：0
作者：
Lijiao You
Di Zhang
Huan Geng
Fangyuan Sun
Ming Lei
机构：
[1] Seventh People’s Hospital of Shanghai University of TCM,Department of Critical Care Medicine
[2] Seventh People’s Hospital of Shanghai University of TCM,Department of Rehabilitation Medicine
来源：
BMC Complementary Medicine and Therapies | / 21卷
关键词：
Sepsis; Salidroside (SAL); Human umbilical vein endothelial cells (HUVECs); Lipopolysaccharide (LPS); Apoptosis; Inflammation; NOD-like receptor protein 3 (NLRP3); Autophagy;
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