Goldmann applanation tonometry compared with corneal-compensated intraocular pressure in the evaluation of primary open-angle Glaucoma

[1] Hollows FC(1966)Intra-ocular pressure, glaucoma, and glaucoma suspects in a defined population Br J Ophthalmol 50 570-586

[2] Graham PA(1980)Biostatistical analysis of the collaborative glaucoma study. I. Summary report of the risk factors for glaucomatous visual-field defects Arch Ophthalmol 98 2163-2171

[3] Armaly MF(1999)The ocular hypertension treatment study: design and baseline description of the participants Arch Ophthalmol 117 573-583

[4] Krueger DE(1857)Amaurose mit sehnervenexcavation Arch. Ophthalmol 3 484-489

[5] Maunder L(1976)Low-tension glaucoma Am J Ophthalmol 81 761-767

[6] Becker B(1986)Comparison of visual field defects in normal-tension glaucoma and high-tension glaucoma Am J Ophthalmol 102 402-404

[7] Hetherington J(2002)The Ocular Hypertension Treatment Study: a randomized trial determines that topical ocular hypotensive medication delays or prevents the onset of primary open-angle glaucoma Arch Ophthalmol 120 701-713

[8] Kolker AE(2003)Factors for glaucoma progression and the effect of treatment: the early manifest glaucoma trial Arch Ophthalmol 121 48-56

[9] Levene RZ(2000)7. The relationship between control of intraocular pressure and visual field deterioration.The AGIS Investigators Am J Ophthalmol 130 429-440

[10] Maumenee AE(1998)Comparison of Glaucomatous Progression Between Untrested Patients with Normal-Tension Glaucoma and Patients with Terapeutically Reduced Intraocular Pressures Am J Ophthalmol 126 487-497

[1] Hollows FC(1966)Intra-ocular pressure, glaucoma, and glaucoma suspects in a defined population Br J Ophthalmol 50 570-586

[2] Graham PA(1980)Biostatistical analysis of the collaborative glaucoma study. I. Summary report of the risk factors for glaucomatous visual-field defects Arch Ophthalmol 98 2163-2171

[3] Armaly MF(1999)The ocular hypertension treatment study: design and baseline description of the participants Arch Ophthalmol 117 573-583

[4] Krueger DE(1857)Amaurose mit sehnervenexcavation Arch. Ophthalmol 3 484-489

[5] Maunder L(1976)Low-tension glaucoma Am J Ophthalmol 81 761-767

[6] Becker B(1986)Comparison of visual field defects in normal-tension glaucoma and high-tension glaucoma Am J Ophthalmol 102 402-404

[7] Hetherington J(2002)The Ocular Hypertension Treatment Study: a randomized trial determines that topical ocular hypotensive medication delays or prevents the onset of primary open-angle glaucoma Arch Ophthalmol 120 701-713

[8] Kolker AE(2003)Factors for glaucoma progression and the effect of treatment: the early manifest glaucoma trial Arch Ophthalmol 121 48-56

[9] Levene RZ(2000)7. The relationship between control of intraocular pressure and visual field deterioration.The AGIS Investigators Am J Ophthalmol 130 429-440

[10] Maumenee AE(1998)Comparison of Glaucomatous Progression Between Untrested Patients with Normal-Tension Glaucoma and Patients with Terapeutically Reduced Intraocular Pressures Am J Ophthalmol 126 487-497